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21 Cards in this Set

  • Front
  • Back
atrio-ventricular ring
support structure for mitral and tricuspid

insulates atria and ventricles from electric signals - means only electrical activity can go down the his bundle
sinus node dysfunction
persistent bradycardia

weakness, fatigue, decrease exercise tolerance

see sudden pauses - greater than 3 seconds -> syncope (stokes-adams attacks)
electrical conduction system of the heart
SA node -> atrial muscle -> AV node -> bundle of his

bundle of his -> RBBB + LBBB

LBBB -> Left posterior fascicle + left anterior fascicle
AV node
acts like a filter - for atrial conductions to pass through to venticles

slow, decremental conduction
Ca++ channel dependent
left bundle branch block conduction pathway
damage in the left bundle branch prevents signals from passing through

electrical conduction must got through RBB conduct through the RV and into the LV

widened QRS complex
right bundle branch block conduction pathway
must conduct through LBB -> septum -> LV -> RV
causes of his bundle/bundle branch blocks
ischemia

calcific valvular disease
causes of AV node dysfunction
ischemia

congenital

iatrogenic

drugs

neural mediated

normal/athletes
2nd degree AV block Mobitz type 1 - site of dysfunction in conduction system
likely in AV node if normal QRS

if BBB then it can be in AV node or in His bundle or bundle branch
2nd degree AV block Mobitz type 2 - site of dysfunction in conduction system
likely below the AV node in distal conduction system

risk of complete heart block
paroxysmal supraventricular tachycardia
tachycardia induced through an abnormal electrical pathway

the abnormal pathways allow signals sent down the conduction pathway to pass through the ventricles and back up into the atria and to the AV node

creating a cycle of conduction

doesn't stop until there is a refractory state that breaks the cycle - can use medications to increases refractory state of AV node (verapamil, adenosine)

can use catheter ablation to destroy the abnormal conducting pathway
atrial fibrillation vs flutter
both don't see p-waves

atrial fib - irregularly irregular - chaotic

flutter - has a repeating sawtooth pattern - more organized
treatment of afib
slow ventricular rate - diltiazem (watch for bradycardia after sinus conversion)

block AV node - digitalis, b-blocker, ca-blocker

terminate the attack - quinidine, flecainide, aniodarone, ibutilide, (anti-arrhythmics)

shock
afib complications
STROKE - clot formation - embolism

put afib patients on coumadin
ventricular tachycardia/ventricular fibrillation
VERY BAD

need shock

anti-arryhthmic drugs - lidocaine, amiodarone

cardiac compressions, IV epinephrine
hypertensive heart
weight > 500g

LV thickeness > 2cm

concentric thickening - reduction in chamber size

microscopic - boxcar nuclei in enlarged myocytes, interstitial fibrosis
cardiac hypertrophy
increased work -> increased size/wall thickness

eventually with decompensate due to volume overload -> dilation and stretching (dialated hypertensive heart)

risk factor for sudden death, arrhythmias, aortic aneurysm, cerebral infarct
left sided heart failure
will see effects of right heart failure as right sided heart failure is caused by left sided failure
right sided heart failure
rare in isolation

its caused by left sided heart failure most common

cor pulmonale "heart lung" - increased resistance of blood flow through lung -> right side hypertrophy -> failure
cor pulmonale
pulmonary fibrosis or emphysema -> increased resistance through pulmonary vessels -> right sided hypertrophy -> failure
T/F atrial diatation can cause atrial fibrillation
true