• Shuffle
    Toggle On
    Toggle Off
  • Alphabetize
    Toggle On
    Toggle Off
  • Front First
    Toggle On
    Toggle Off
  • Both Sides
    Toggle On
    Toggle Off
  • Read
    Toggle On
    Toggle Off
Reading...
Front

Card Range To Study

through

image

Play button

image

Play button

image

Progress

1/29

Click to flip

Use LEFT and RIGHT arrow keys to navigate between flashcards;

Use UP and DOWN arrow keys to flip the card;

H to show hint;

A reads text to speech;

29 Cards in this Set

  • Front
  • Back

What do cranial nerves do?

Supply head with all of the functions supplied to the body by spinal nerves




Include specialized functions such as vision, taste, hearing, and balance and autonomic functions.

How are cranial nerves tested ?

Functional manipulation designed to elicit a deficit specific to a particular nerve.




Tested in conjunction with symptoms

Why can PN and CN lesions involved one or more nerves?

Their close proximity in the brain or brainstem and their and their peripheral pathways

What can loss of motor function of CN result from?

Loss of central tracts that control motor functions (UMN)




Loss of motor neurons themselves or their axons (LMN)

What is an UMN lesion?

All motor system lesions that involve neurons or tracts that are not directly connected to muscles

What is a LMN lesion?

Involve neurons or their axons that directly contact muscle

What are typical LMN symptoms?

Flaccid Paralysis - relatively rapidly by atrophy of the muscle




Fibrillations of motor units as a result of denervation




Hypotonia - Decreased muscle tone




Hypotreflexia/areflexia - weakening or absence of tendon reflexes

What occurs from LMN decifits

Deficits are permanent, although if incomplete, some compensation/recovery can occur




If functional loss results from more distal lesions of axons in a nerve, some limited functional regeneration of axons may be possible

What is UMN syndrome?

result of denervation of motor neurons. A typical cause is stroke or other vascular occlusion in the brain or brainstem.




The closer the lesion is to the origin of the tract, that all motor functions below the lesion are lost.

What are symptoms of UMN Lesions

Weak and flaccid muscles followed by spasticity, hypertonia, hyperreflexia, clonus and altered cutaneous reflexes.

Describe recovery from UMN lesions?

Recovery is more likely with cranial nerve motor nuclei because the motor cortex from both hemispheres usually innervates motor neurons on both sides

How do brain lesions typically present initially?

Lesion on one side of the brain presents as initial weakenss on the contralateral side followed by recovery. Some disturbances of reflexes may occur where they can be examined

Describe the trigeminal nerve innervation

Sensation to the face wtih the back of the head coered by C2-C3. CN5 carreis both epicritic and protopathic types of sensation

Describe lesions of epicritic systems?

Will produce a loss or reduction in two point discrimination, the ability to feel vibration and the ability to tell the position of abody part in the absence of visual confirmation

Two point vs Kinesthetic testing?

CN 5, 2pt Discrimination and vibration can be tested but Kin cannot

Describe lesions of the anterolateral system

Produce contralateral loss of protopathic touch, pain, and temperature


-pain is usually detected with a pin


-Temp with tubes of cold and warm water



How are Cranial nerves 9 and 10 used to evaluated sensory info

Usually done by triggering reflexes, such as gag and cough. BP and HR changes can also be examined, although these are not typically used to test CN functions

Cranial Nerve 1 Deficit

Olfactory - Smell


D: Anosmia




Occludes one nostril, use an aromatic substance. ID Odor.

Cranial Nerve 2 deficit?

Optic - vision


D: Anopsia




Check eyes individually for ability to detect objects in the visual field

Cranial Nerve 3 deficit

Oculomotor




Diplopia - double vision, external strabismus, loss of parallel gaze, ptosis




Dilated pupil, loss of motor limb of light relfex with CN 2




Eye deviates out and down due to unopposed actions of remained muscles

Cranial Nerve 4 deficit

Weakness looking down when eye is abducted,




Difficulty reading, going down stairs




Head may tilt away from the lesion side

Cranial Nerve 5 deficit

Trigeminal




Loss of sensation in skin of forehead


Loss of sensory limb of blink reflex


Loss of sensation over maxilla


Loss of sensation in skin over mandible




Weakness in chewing, jaw deviation toward lesioned nerve


Decreased or absent jaw jerk reflex

Cranial Nerve 6 deficit

Abducens




Diplopia, internal strabismus, loss of lateral gaze

Cranial nerve 7 deficit

Facial




Corner of mouth droops, cannot close eye, loss of blink reflex




Hyperacusis


Alteration or loss of sense of taste


Reduction in saliva output


Dry eye


Reduction in secretion

Cranial nerve 8 deficit

Vestibulocochlear




Sensorineural hearing loss


Loss of balance, vertigo, nystagmus

Cranial Nerve 9 deficit

Glossopharyngeal




Loss of gag reflex, sinus reflex


Sensory loss on posterior tongue


reduction in salivation


Swallowing difficulty


Loss of taste to posterior tongue

Cranial nerve 10 deficit

Vagus




Palate droop, uvula pointing away from lesioned nerve


Dysphagia, loss of gag reflex


Hoarseness, dysphonia

Cranial nerve 11 Deficits

Spinal Accessory




Weakness turning head to opposite side


Shoulder droop, difficulty combing hair

Cranial nerve 12 deficit

Hypoglossal




Tongue deviation on protrusion toward lesioned nerve




Dysarthria (Difficulty speaking)