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70 Cards in this Set

  • Front
  • Back
what does upper GI series visualize?
esophagus, stomach, duodenum
area gastricae vs rugal folds
both found in stomach; area gastricae are more fine mosaic pattern, primarily in gastric antrum
when to use gastrograffin vs barium?
gastrograffin only if emergency (eg ?perf); gastrograffin is water soluble, secreted by kidneys, so no big deal if extravasates; also, gastrograffin doesn’t require prep (unlike NPO after midnight for barium)
when not to use gastrograffin?
can cause pulmonary edema --> contraindicated if aspiration risk
esophageal anatomy on barium swallow: 3 segments, 2 lines
SEGMENTS: tubular (majority), vestibular (bulb like area near GEJ), and submerged segments (leads to GEJ); LINES: A line (b/w tubular and vestibular portions); Z line / B ring: between vestibular and submerged segments, represents squamocolumnar junction
types of peristalsis (3)
1) PRIMARY: stripping wave init by swallowing; 2) SECONDARY: stripping wave follows primary, gets remaining food; 3) TERTIARY (nonstripping, disordered)
what nerve controls esophageal motility? Where is the nucleus?
Vagus nerve (dorsal vagal nucleus in the midbrain)
causes of primary esophageal dysmotility
achalasia, diffuse esophageal spasm, nutcracker esophagus, others
causes of secondary esophageal dysmotility
1) MIDBRAIN: MS, CVA; 2) VAGUS: esoph/lung cancer; 3) MYENTERIC/MESENTERIC PLEXI: esophagitis, esophageal CA, scleroderma, neuropathy
classic radiographic features of achalasia (4)
1) absent GASTRIC BUBBLE; 2) early --> disordered peristalsis, late --> absent peristalsis; 3) Dilated esophagus with AIR FLUID level; 4) BIRD BEAK lower esophagus
what to r/o if ?achalasia?
tumor (and other 2' causes of achalasia), incl esophageal and lung CA
what parts of esophagus affected by scleroderma?
distal 2/3 --> atony
ddx of air in a dilated esophagus (3)
1) scleroderma; 2) Achalasia; 3) lower esophageal stricture
what 3 conditions cause absent peristalsis?
1) LATE achalasia; 2) Scleroderma; 3) Neuropathy (DM, EtOH)
Types of hiatal hernias (3)
TYPE 1: Sliding (95%); TYPE 2: Paraesophageal (5%); Type 3: Combination (rare)
how to dx a small sliding hiatal hernia?
B-line > 1cm above hiatus between swallows (during swallows, submerged segment submerges below hiatus)
what's seen with sliding hiatus hernia on CXR?
retrocardiac density
what is bochdalek hernia?
persistent fetal posterior pleuroperitoneal canals -- > allows herniation of abdominal contents (usu L sided)
pulsion vs traction esophageal diverticulae
PULSION: a/w disordered peristalsis: Zencker's (posterior) and Epiphrenic (lateral); TRACTION: a/w extrinsic abnormalities (eg tuberculous mediastinal node), usu mid-esophageal
what are feline folds?
fine esophageal folds caused by submucosal contractions 2/2 esophagitis / irritation
what does esophageal cancer look like on barium swallow?
apple-core appearance
what does post-radiation esophageal stricture look like?
smooth tapered edges with stricture in the middle --> looks benign ("rat tail")
what does barrett's esophagus look like on barium swallow?
mid-esophageal stricture with reticular mucosal pattern, +/- ulcer, +/- hiatal hernia
what to do to work up any stricture?
biopsy! (r/o cancer)
use of single vs double contrast study
single better for polyps, double better for fine mucosal abnormalities
ddx for varicoid lesion on barium swallow? (2)
1) Varices; 2) esophageal tumor (carcinosarcoma / lymphoma)
candidal vs Herpes esophageal ulcers
candidal ulcers more course
what to CMV / HIV esophageal ulcers look like?
EITHER multiple diamond-shaped ulcers with halo OR giant ulcer toward GEJ
major risk factors for esophageal cancer
SMOKING (synergistic with EtOH), Barrett's esophagus, achalasia, scleroderma
what is boerhaave's syndrome
RUPTURE OF ESOPHAGUS: linear tear across GEJ 2/2 vomiting across closed glottis (often in drunk/passed out); similar to mallory-weiss, but goes THROUGH the wall, not just mucosal tear ==> no hematemesis, but aberrent air seen on cxr
thickness of esophagus on CT
<3mm
stomach visualization in UGI with prone vs supine positions
SUPINE: fundus and antrum are posterior --> fill with contrast; PRONE: body is anterior, so fills with contrast
differences between benign and malignant ulcers: (7)
1) PROJECTION (outside vs inside lumen); 2) BORDER (smooth vs irregular); 3) LOCATION relative to edema (central vs eccentric); 4) RADIATING FOLDS (start close to vs far from crater edge); 5) RADIATING FOLD THICKNESS (uniform vs clubbed); 6) hampton's line vs carmen kirklin complex; 7) HEALING (complete vs incomplete)
what is a hamptom's line?
thin, sharply demarcated radiolucent line along the neck of a contrast-filled BENIGN gastric ulcer, indicates mucosal edema
what is the carmen kirklin complex?
pathognomonic of malignant gastric ulcer: push on epigastrium --> see shadow?
what is linitis plastica?
infiltrating scirrhous carcinoma ==> extensive thickening of stomach wall (aka leather-bottle stomach)
causes of linitis plastica
infiltration: malignancy, granulomatous, radiation changes, benign ulcers
what causes thickened mucosal folds in the stomach?
lymphoma, menetrier's dz, gastritis, varices (proximal)
where is the ligament of treitz?
at the duodenal-jejunal junction
rule of thumb for duodenal ulcers
benign until proven otherwise
what does cyst look like on US?
anechoic collection + increased distal intensity
what causes duodenal hematoma?
often MVA (esp C3 segment)
what can a pancreatic pseudocyst do to duodenum?
cause c-loop widening (sweep abnormality)
rule of thumbs for duodenal vs gastric ulcers
duodenal benign until proven otherwise -- usu no endoscopy; gastric can be either benign or malignant, radiology helps, but most are endoscopied / biopsied (if any suspicious features)
three spaces around kidney used on CT
anterior pararenal space, perirenal space, posterior pararenal space
ionic vs nonionic contrast
ionic has fewer complications a/w it
contraindications to IVU (intravenous uretrogram)
same as contraindications to contrast
3 phases of IV contrast injection (in reference to kidneys)
1) total body opacification (kidney and liver equally opacified); 2) nephrogram phase (cortex lit up); 3) pyelogram phase (within 3-4mins --> contrast in renal calyces, pelvices, ureters)
3 stages of reading a IVU
1) SCOUT FILM: radio opacities/lucencies; 2) NEPHROGRAM PHASE (renal fxn, parenchymal morphology); 3) PYELOGRAM PHASE (timing, tubular evaluation -- polyps, fistulas, etc)
contraindications to contrast (6)
1) PREGNANCY; 2) ALLERGY to IV contrast; 3) RENAL INSUFFICIENCY; 4) MULTIPLE MYELOMA; 5) CHF; 6) GOUT
uses of ultrasound in renal evaluation
1) renal mass vs cyst; 2) doppler eval of blood flow; 3) guide biopsies / drainage
what is a bosniak type I renal cyst?
SIMPLE cyst: fluid filled, no calcification or wall thickening; BENIGN
what is bosniak type II cyst?
slightly more complex than type I: homogenous cyst with 1-2 septations, nonenhancing; BENIGN
what is bosniak type IIf cyst?
COMPLEX cyst: some suspicious features warranting followup, eg enhancing rim; PROBABLY BENIGN (but needs followup)
what is bosniak type III renal cyst?
COMPLEX cyst: >2 septations, ?enhancing septations, wall thickening, nodularity; POSSIBLY MALIGNANT
what is bosniak type IV renal cyst?
usu due to renal carcinoma with cystic component; MALIGNANT
classic findings on different imaging modalities of renal cyst
anechoic on US with enhanced through-transmission, no uptake on CT, hypodense on T1, hyperdense on T2
Robson staging of renal cancers
1: inside capsule; 2: outside capsule, inside fascia; 3: nodes/vein involvement; 4: outside fascia/distant sites
stone vs polyp in ureter
stone causes spasm --> stenosis; polyp causes Bergman's sign (uretral dilatation distal to obstruction
what renal pathology can be confidently diagnosed on US/CT/MRI?
angiomyolipma -- contains bulk fat --> hyperechoic on US, in phase and out of phase appearnace on MRI, fatty appearance? on CT
weigert-meyer law
in duplex kidney, upper pole obstructs, lower pole refluxes
what is renal scan good for?
differentiating obstructive vs nonobstructive hydronephrosis, functional studies (gfr), HTN, infection
which stones radioopaque on CT?
ALL! (except some anti-retroviral- associated stones, eg indinivir)
what does pyelonephritis look like on CT?
wedge-shaped area of low attenuation
differentiating b/w ACUTE pyelonephritis and ACUTE infarct on CT
both show wedge-shaped hypodensity, but in pyelonephritis, wedge is pointed TOWARD calyx, while in infarct, wedge is pointed BETWEEN calyces
differentiating b/w CHRONIC pyelonephritis and CHRONIC infarct on CT
both show wedged-shaped defect of PARENCHYMA, but pyelo --> apex of defect is TOWARD CLUBBED calyx, while infarct --> apex BETWEEN NORMAL calyces
3 MCC papillary necrosis
1) ANALGESIC ABUSE; 2) DM; 3) SICKLE CELL DISEASE
calyceal clubbing seen with what?
seen with both papillary necrosis and hydronephrosis; in hydronephrosis, also see pelvicalyceal dilation
3 manifestations of renal osteodystrophy
1) diffuse osteosclerosis; 2) osteomalacia; 3) 2' hyperPTH
findings in 2' hyperPTH (3)
distal resorption of b/l clavicles; widening of SI joints and pubic symphysis; horizontal linear lucency through centers of multiple vertebral bodies ("rugger jersey spine")