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DHP receptor on T-tubule transmits this info to Ryanodine receptor on surface of sarcoplasmic reticulum.
Ryrs causes Ca2+ to be sequestered from SR and Ca2+ leaves through Ryrs and released from terminal cisternae of SR and then binds to troponin... (see L06)
How can we treat it?
It can be treated by AChE inhibitors such as neostigmine as they prolong the time ACh lingers so there is an increased chance of binding to receptors for muscle contraction.
- Inability to smile
- Saggy face (as we need lots of muscles to keep face)
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