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18 Cards in this Set

  • Front
  • Back
Chronic Obstructive Pulmonary Disease
-Umbrella term for emphysema and chronic bronchitis (usually one predominates)
-Results from repeated exposure to respiratory irritants that damage structure of lungs
-Irreversible airflow limitation on exhalation
-Over 15 million Americans diagnose with COPD
-4th leading cause of death in the US
-Is not CURABLE, but it can be managed
-Direct cost of care estimated at 18 billion
Risk Factors
-#1 risk factor tobacco smoking
-Occupational exposure
Environmental pollution
-Recurrent respiratory infections
-Hereditary -Alpha-antitrypsin (AAT) deficiency
-Clients with asthma
-Aging process
Emphysema
-Airflow limitation is the result of lung tissue destruction
-Destruction of the alveolar walls > outpouring of macrophages digest alveolar tissue > decrease surface area for diffusion
-Destruction of elastin > loss of elastic recoil > hyperinflation of lungs > airways collapse on expiration > air trapping in alveoli
S&S Emphysema
Progressive dyspnea
Prolonged expiratory phase
Barrel chest
Use of accessory muscles to breathe, assumes tripod position
Diminished breath sounds
Weight loss/underweight
Clubbing
Hypoxemia/hypercapnia develop later in the disease process
Chronic Bronchitis
(productive cough x 3 months in 2 consecutive years)
-Chronic inflammation of the airways with vasodilation, congestion and edema of mucosa
-Mucosal inflammation edema  further narrowing of the airway
-Thick & tenacious mucus in airways  bacterial medium  ↑ susceptibility to infection
-Narrowed airways and excess secretions obstruct airflow
S&S Chronic Bronchitis
Dyspnea
Persistent, productive cough (white)
Bronchospasms at the end of coughing episodes
Wheezing/crackles
Prolonged expiration
Polycythemia
Ruddy appearance/Cyanosis
Complications of COPD
-Cor pulmonale/right sided hear failure
-Acute exacerbations of COPD
-Pneumonia
-Respiratory failure
-GERD/ulcers
-Depression
Cor Pulmonale
-hypertrophy of right side of heart
-result of pulmonary hypertension secondary to hypoxia and hypercapnia
-Eventually will cause right sided heart failure
Clinical Manifestations of right sided heart failure
-Peripheral edema
-Weight gain
-Ascites
-Hepatomegaly
-Bounding pulses
-JVD
-Changes in heart sounds and ECG
Diagnostic Testing
History
Clinical presentation
Spirometry/PFT
ABG’s
CXR
Sputum Gram stain, C&S
AAT deficiency serum testing
Collaborative Therapy
Cessation of smoking (wellbutrin, chantix)
Treatment of exacerbations and resp. infections
Drug therapy
Bronchodilators - nebulizer, inhalers, pills
Cortiocsteroids- iv push/piggyback solumedrol 40-100 mg, then switched to pregnesone inhaled- know how to admin dpi vs mdi for exam!
Expectorants
Oxygen therapy- lowest possible level, goal of o2 sat of 90% or greater
Chest phsyiotherapy
Nutrition- issues w weightloss, hi cal and hi protien diets, low carbs
Exercise- 20 - 30 min 3x a week
Pulmonary rehabilitation
Vaccinations - pneumonia and flu
Surgical Therapy
Lung volume reduction surgery
-remove 30% of disease lung tissue

Bullectomy
-used for emphysematous COPD

Lung transplantation
Asthma Statistics
-Asthma affects more than 20 million adults in the US
-Asthma is the most chronic disease of childhood; most common cause of hospitalization for children under 15. Over 9 million children have asthma
-Estimated that 80% of asthma deaths could be prevented with proper asthma education.
Asthma is attributed to:
-Nearly 2 million ER visits,
-25 million lost work days annually
-15 million lost school days annually
-Health care costs over $16 billion a year
-If 50% of asthmatic adults and children received the flu vaccine then $379 million could be saved in hospital costs.
-Currently, fewer than 10% of children and 40% of adults with asthma are vaccinated.
Asthma
-Chronic inflammatory disease of the airways, alveoli not involved
-Intermittent, reversible airflow limitation
Triggers of Asthma
Allergens
Air Pollution
Occupational Exposure
Respiratory Infections
Exercise
Drug/Food Additives
GERD
Nose/Sinus Problems
Stress
Asthma PATHOPHYSIOLOGY
-Chronic inflammation from exposure to allergens or irritants
-The airways are hyperresponsive which lead to airway obstruction from overproduction of mucus and edema of the ariway mucosa
Inflammatory mediators cause
Vascular congestion
-Edema formation
-Production of thick, tenacious sputum
-Bronchoconstriction
-Thickening of airway walls
-Increase bronchial hyperresponsiveness
Asthma Clinical manifestations
-Cough
-Wheezing
-Dyspnea
-Chest tightness
-Tachycardia with tachypnea
-Prolonged expiratory phase
-Anxiety/restlessness
-Use of accessory muscle
retractions