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8 Cards in this Set
- Front
- Back
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Lesion study = causation |
fMRI = correlations |
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We see deficits to the DLPFC and ACC in drug users, but we also see this deficit in problem-gamblers. What does this suggest? |
That perhaps "drugs" do not cause these deficits, but if perhaps they are caused by something else that makes these people vulnerable to addition |
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What are some studies that are under way that are looking at pre-morbid risk in teenagers, that can predict whether naive children of drug users will then go on to become drug users? |
1. Walter Michel studies on delayed gratification 2. Longitudinal studies of brain images in individuals (e.g. predicting binge drinking in a sample of 2000 young adults in Europe) |
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What is the limitation of a large cohort study? |
Not very good at assessing the rare things, such as dependency on methamphetimines. Would have to wait a long time for these things to emerge |
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What is another way to assess the cognitive abilities of a drug user for predictive purposes? |
The emotional Stroop task. Start no words, neutral words, category words, emotionally laden words |
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What is the point of doing all these cognitive performance tests on drug users? |
to try and match people to effective treatment plans, but cognitive assessments don't have great predictive power. |
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Is there cognitive recovery in methamphetamine users if they abstain? |
Not really. Relapsers are generally more cognitively able than abstainers, but over time the abstainers become faster at motor skills and processing, but not for memory and learning. But we can give people ritalin to improve cognitive performance on tasks |
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So why don't be give MA users when detoxing drugs to help them get better? |
Systematic reviews have found that they aren't effective, really, for preventing relapse. They may be effective in the initial withdrawal stage, to help them be cognitively perk in order for them to participate in psychological treatments, such as CBT |
