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22 Cards in this Set

  • Front
  • Back

Upper Motor Neuron Disease

lesion found in descending motor tracts within the cerebral motor cortex, brain stem or spinal cord.




S/S: weakness, hypertonicity, hyperreflexia, abnormal reflexes




Ex: CP, ALS, CVA, birth injuries, MS, TBI, brain tumors

Lower Motor Neuron Disease

lesion affects the nerves or axons at or below level of brain stem




S/S: flaccidity, decreased tone, atrophy, absent reflexes




Ex: ALS, Guilliain-Barre syndrome, trauma, infection, Bell's palsy, carpal tunnel syndrome, MD

Aphasia

acquired neurological impairment of processing for receptive or expressive language.

Alzheimer's Disease

progressive neurodegenerative disorder, irreversible damage in cerebral cortex and subcortical areas of brain.




S/S: difficulty with learning new things, loss of orientation, word-finding difficulties, depression, poor judgement, shuffling gait, dependent, inability to speak




Tx: max pt learning and maintaining current function, caregiver edu

Amyotrophic lateral Sclerosis (ALS)

chronic degenerative disease that has both UMN and LMN impairments




S/S: LMN - asymmetric muscle weakness, fasciculations, cramping and atrophy in the hands. UMN - incoordination, spasticiy, clonus and positive babinski reflex. fatigue, paralysis and respiratory paralysis




Tx: supportive care and quality of life

Carpal Tunnel Syndrome

peripheral nerve entrapment injury that results of compression of median N.




S/S: sensory changes and paresthesia, can radiate into UE, shoulder and neck, night pain, weakness of hand, atrophy, decreased grip strength, and clumsiness




Tx: splinting, ergonomic measures, corticosteroid injections and PT. surgery is possible.

Congenital Malformations

happen early in life and are non-progressive. vary depending on structure involved. ataxia is usually present

Hereditary Ataxias

gait unsteadiness early in life followed by UE ataxia, dysarthria and paresis. Mental function declines and tremors begin.

Spinocerebellar Ataxis

affect CNS and PNS, present with neuropathy pyramidal signs, ataxia and restless leg syndrome

Acquired ataxis

aquired from an external source like toxins or may be idiopathic

Diabetic Neuropathy

S/S: weakness and sensory disturbances occur distally and symmetrically. tingling, numbness/pain, wasting of muscles, orthostatic hypotension, weakness, and urinary impairments




Tx: strict monitorying of blood glucose levels to prevent further nerve pathology. pain management, foot care, and overall fitness. medications can be used as well

Epilepsy

chronic condition with temporary dysfunction of the brain causing an electrical discharge and seizure activity




S/S: loss of awareness, sensation, mood or mental function occurs




Tx: antiepileptic meds and surgery is possible

Guillain-Barre Syndrome

acute polyneuropathy, temp inflammation/demyelination of peripheral nerves myelin sheaths.




S/S: motor weakness, sensory impairment, respiratory paralysis. can be life threatening




Tx: hospitalization for symptoms. Meds: immunosupressive, and narcotic meds. Cardiac monitoring and ventilation may be possible. PT: pulmonary rehab, strengthening, mobility training, w/c and AD training.

Huntington's Disease

neurological disorder of CNS - degeneration of basal ganglia and cerebral cortex




S/S: cognitive impairment. involuntary movements, alteration in personality, grimacing, ataxia, decrease in IQ, depression, incontinence, immobility in later stages.




Tx: maximize endurance, strength, balance, postural control and functional mobility.

Multiple Sclerosis (MS)

patches of demyelination of myelin sheaths. decreases efficiency of nerve impulse




S/S: visual problems, paresthesis & sensory changes, clumsiness, weakness, balance dysfunction and fatigue. periods of exacerbation and remission




Tx: meds. PT - regulation of activity level, relaxation and energy conservation techniques,normalization of tone, balance and gait training, core stabilization and AD training.

Myasthenia Gravis

autoimmune disease resulting in neuromuscular junction pathology. antibodies block/destory receptors




S/S: extreme fatigue and muscle weakness, ocular muscles affected first, dysphagia and cranial nerve weakness are common




Tx: will have remissions and exacerbations. PT - energy conservation, strengthening with isometrics

Parkinson's Disease

degenerative disorder with a decreased production of dopamine




S/S: resting tremors in hands or feet that increase with stress, balance disturbances, difficulty rolling over, impairment iwth fine movements, progression includes - sluggish movements, hard to start and stop movements, shuffling gait




Tx: alot of meds: dopamine replacement therapy. PT - maximizing endurance, strength and functional mobility, verbal cueing and visual feedback are effective.

Post-Polio Syndrome (PPS)

LMN pathology that affects anterior horn cells with those who have been affected with polio.




S/S: slow/progressive weakness, fatigue, muscle atrophy, pain and swallowing issues




Tx: lifestyle modification and symptomatic intervention. PT- supervised exercise, functional independence, adaptive equipment and education.

Cerebral Palsy (CP)

movement disorders due to brain damage that are acquired in utero, during birth or infancy.




S/S: abnormal muscle tone, impaired modulation of movement, abnormal reflexes and impaired mobility




Tx: caregiver edu., normalization of tone, stretching, strengthening, positioning, WB activities, mobility skills, splinting, AD, surgery can be possible

Down Syndrome

genetic abnormality of an extra 21st chromosome




S/S: mental retardation, hypotonia, joint hypermobility, flattened nasal bridge, small mouth, flat feet, scoliosis, congenital heart disease




Tx: exercise and fitness, stability, maximizing respiratory function, edu. for caregivers, surgery for cardiac abnormalities.

Duchenne Muscular Dystrophy

progressive disorder casued by absence of gene required to produce muscle proteins




S/S: progressive weakness, disinterest in running, falling, toe walking, excessive lordosis




Tx: caregiver edu, respiratory function, submax exercise, mobility skills, splinting, orthotics and AD

Spina Bifida

insufficient closure of neural tube, occurs usually in low thoracic, lumbar or sacral regions




S/S: motor loss, sensory deficits, hydrocephalus, clubfoot, scoliosis, bowel and bladder dysfunction and learning disabilities




Tx: ppositioning, handling, ROM, ther ex. skin care, strengthening, balance/mobility training, adaptive equipment, splinting, w/c.