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51 Cards in this Set

  • Front
  • Back

ICA branches

Ophthalmic Artery and posterior communicating artery

ICA terminates

In middle cerebral artery and anterior cerebral artery

ECA branches

8 major branches


1st superior thyroid artery


Facial artery, superficial temporal artery

Vertebral Arteries form

Basilar artery

Basilar artery divides inti

Posterior cerebral arteries

Circle of Willis

Largest intra-arterial connection

Vertebrobasilar system

Supraorbital artery arises from

Ophthalmic Artery and joins with ECA via some of it's branches

Frontal artery arises from

Ophthalmic Artery; exits orbits medially to supply mid forehead;joins ECA via some of its branches

Anastomoses include

ECA-ICA via orbital and ophthalmic arteries


Occipital branch of ECA w/ Atlantic branch of vertebral


Bernoulli principle

Total fluid energy along a streamline of fluid flow is constant

Transient ischemic attack

Fleeting neurological dysfunction;symptoms last <24 hrs;usually embolic from heart or carotid artery

Resolving ischemic neurologic deficit

(RIND) symptoms last >24hrs; complete recover usually occurs

Cerebrovascular accident

(CVA) symptoms last >24hrs; complete recovery does not occur

Atheromatous

Form of arteriosclerosis; localized accumulations

Types of atheromatous

Fatty streak:thin layer in intimal


Fibrous plaque


Complicated lesion


Ulcerative lesion: deterioration of normally smooth surface; may result in emboli


Intra-plaque hemorrhage: sonolucent area w/in plaque


Types of atheromatous

Fatty streak:thin layer in intimal


Fibrous plaque


Complicated lesion


Ulcerative lesion: deterioration of normally smooth surface; may result in emboli


Intra-plaque hemorrhage: sonolucent area w/in plaque


Thromboembolic

Obstruction of blood vessel by piece of thrombus

Thrombus

Large amounts of RBC's trapped w/in fibrin network

Embolism

Piece of thrombus breaks loose and travels distally until lodges in small vessel

Aneurysm

Localized dilation of blood vessel due to congenital defects or weakness of wall (trauma, infection or atherosclerosis)

Dissection

Results from sudden tear in intima; creates false lumen

Fibromuscular dysplasia

(FMD) most commonly caused by dysplasia of media along w/ overgrowth of collagen in mid/dist ICA; bead like appearance;often seen in young women

Neointimal hyperplasia

Intimal thickening from rapid production of smooth muscle cells; response to vascular injury/reconstruction (endarterectomy);significant stenosis may occur w/in 6-24 months

Symptoms w/ ICA lesions

Unilateral paresis, unilateral paresthesia, aphasia, amaurosis fugax

Symptoms w/ MCA lesions

Aphasia or dysphasia, more severe facial and arm hemiparesis or hemiplegia, behavioral changes

Symptoms w/ ACA lesions

More severe leg hemiparesis or hemiplegia, incontinence, loss of coordination

Paresis

Weakness or slight paralysis on one side of body

Paresthesia

Prickling or tingling of skin

Aphasia

Inability to speak

Amaurosis fugax

Temporary, partial or total blindness usually of one eye

Dysphasia

Impairment of ability to communicate

Hemiparesis

Weakness on one side of body

Hemiplegia

Paralysis on one side of body

Symptoms w/ vertebrobasilar lesion

Vertigo, ataxia, bilateral visual blurring or double vision, bilateral paresthesia or anesthesia, drop attack

Symptoms w/ PCA lesion

Dyslexia, coma

Ataxia

Muscular uncoordination

ICA Doppler signal

High pitched and continuous; rapid upstroke and down stroke with high diastolic component

ECA doppler signal

Pulsatile; rapid upstroke and down stroke with low flow diastole, dicrotic notch

50-79% stenosis

PSV >125


EDV <140

80-99% stenosis

PSV >125


EDV >140

Surgery w/ ICA/CCA ratio

>4

Criteria determining occlusion

CCA mat have very low or absent diastolic flow; evidence of collateralization (ECA exhibit high flow in end diastole); absent ICA Doppler signal or pre-occlusive thump

Ways to increase PRF/Nyquist limit

Decrease baseline; increase scale; change frequency; alter angle; decrease depth; use CW

Mirror imaging

Doppler shifts above and below baseline; turn down gain or change angle of isonation

Helical Flow

Occurs when Flow moves into wider portion of vessel; Doppler shifts above and below baseline; spectral broadening present

Intraoperative monitoring

Identify defects secondary to surgery or areas of platelet aggregation; use >12 MHz transducer

If flow in ACA is antegrade

Getting flow from anterior communicating artery

If increased flow in PCA

Reversing direction of flow in posterior communicating artery

Temporal arteritis

Inflammation of arterial wall

Calculating diameter reduction

(1-(d/D)x100=%