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118 Cards in this Set

  • Front
  • Back
In a tall, slender person, the heart tends to hang more
Vertically and positioned centrally
In a more stocky and short person the heart thends to lie more
To the left and positioned horizontally
The heart may be positioned to the right, either rotated or displaced (mirror image of what is expected)
Dextrocardia
When the heart and stomach are placed to the right and the liver to the left this habitus is termed
situs inversus
Thin outermost muscle layer of the heart that extends onto the great vessels
Epicardium
Primary muscle mass of the heart consists of the
Right and left ventricles
Most of the anterior surface of the heart is formed by the
Right ventricle
Poster to the right ventricle but extends around to the anterior is the
Left Ventricle
Apical impulses caused by the ventricular contraction and thrust are felt here
5th left intercostal space at the midclavicular line
Coronary arteries and veins are found below this layer
Serous pericardium (visceral layer epicardium)
The most posterior aspect of the heart is formed by the
Left atrium
Dimensions of the heart
12 cm long; 8 cm wide at the widest point; 6 cm AP diameter
The only intracardiac pathways of a normal heart
Atrioventricular and semilunar valves
AV valves
Tricuspid and mitral valve
Semilunar vavles
Pulmonic and aortic
Contraction of the ventricles open these valves
Semilunar valves
Ventricle contraction shuts these vavles
AV valves
Phase of cardiac cycle when ventricles contract
Systole
Energy-requiring phase of cardiac cycle when ventricles dilate
Diastole
First heart sound (S1; "lubb") is caused by
AV vavle closure
Usual sound of valves opening
None
Closure of these valves causes the second heart sound (S2, "dubb")
Semilunar vavles
Components of the S2
A2 (aortic valve) and P2 (pulmonic valve)
S3 is a heart sound caused by
Filling of the ventricles
S4 is a heart sound caused by
Ejection of blood from the contraction of the atria
Heart sound heard during isometric contraction
S1
Heart sound heard during isometric relaxation
S2
Cardiac phase of rapid ejection and reduced ejection phase
Systole
P wave represents
Atrial depolarization
Normal PR interval
0.12 - 0.20 secs
PR interval represents
Time from stimulation of the atria to stimulation of the ventricles
QRS complex represents
Ventricular depolarization
Normal interval of QRS complex
< 0.10 secs
T wave represents
Ventricular repolarization
Closure of the ductus arteriosus usually occur within (hrs)
24-48 hrs
Relative size of the adult ventricular sizes
Left is twice as big as right ventricle
Age when ventricular size approximate to that of an adult's?
1 year
Usually adult heart position is rached by this age
7 years
Blood volume increase during pregnancy
40-50%
Time when blood volume returns to normal postpardum (wks)
3-4 weeks
During pregnancy, cardiac output increases by
30-40%
After delivery, cardiac output returns to normal after (wks)
2 weeks
Cardiac output reaches highest level by (wks)
25 to 32 weeks of gestation
Vavles most affected by fibrosis and sclerosis in older adults
Mitral vavle and aortic cusps
Common EKG changes in older patients
1st degree AV block, bundle branch blocks, ST-T wave abnormalities, premature systole (atrial and ventricular), left anterior hemiblock, left ventrcular hypertophy and atrial fibrilation
A sudden, sharp, relatively brief non-radiating pain, occurs most often at rest and is unrelated to exertion and mya not have a discoverable cause
Precordial catch
Possible causes of chest pain
Cardiac
Aortic
Pleuropericardial Pain
GI disease
Pulmonary disease
Musculoskeletal
Psychoneurotic (illicit drug use)
Exercise intensity of getn' it on
Moderately Heavy (climbing 1-2 flights of stairs, lifting full cartons, long walks)
Risk factors for cardiac disability that would increase chances by app. 8x > if none is present
Hyperlipidemia
Smoking
DM
Upon palpation of your patient's PMI, you feel an apical impulse that is more forceful and widely distributed, last throughout systole or is laterally and downwardly displaced which may indicate...
Left ventricular hypertrophy
You feel lift along the left sternal border which may be caused by...
Right ventricular hypertrophy
An apical impulse displaced to the right without a loss or gain in thrust suggests...
dextrocardia, diaphragmatic hernia, distned stomach or a pulmonary abnormality
A fine palpable, rushing vibration or palpable murmur
Thrill
Most likely place to feel a thrill on the chest of your patient
Over the base of the hear in the area of the right or left second intercostal space
A thrill might indicate these problems
Defect in the closure of one of the semilunar valves, pulmonary hypertension or atrial septal defect
Location of the carotid pulse in relation to the jaw
Medial and inferior to the angle of the jaw
Grade level of a thrill
Grade IV of more
Thrills that are found at these sitse during systole may indicate these probable causes:
- Suprasternal notch and/or 2nd & 3rd right rib space
- Suprasternal notch and/or 2nd & 3rd left rib space
-4th left intercostal space
-Apex
-Left lower sternal border
-Left upper sternal border often with extensive radiation
- Suprasternal notch and/or 2nd & 3rd right rib space = Aortic stenosis
- Suprasternal notch and/or 2nd & 3rd left rib space = Pulmonic stenosis
-4th left intercostal space = Ventricular Septal Defect
-Apex = Mitral Regurge
-Left lower sternal border = Tetralogy of Fallot
-Left upper sternal border often with extensive radiation = Patent ductus arteriosus
Thrills that are found at these sitse during diastole may indicate these probable causes:
- Right sternal border
- Apex
- Right sternal border = Aortic Regurgitation or Aneurysm of ascending aorta
- Apex = Mitral stenosis
Amount of pressure when using the diaphragm of your stethoscope, and bell?
Firm pressure with diaphragm
Light pressure with the bell
5 traditionally designated asucultatory areas (whatcha listening to?):
- 2nd right rib space at the sternal border
- 2nd left rib space at the sternal border
- 3rd left rib space at sternal border
- 4th left rib space along sternal border
- 5th left rib space at the MCL
- 2nd right rib space at the sternal border = Aortic valve area
- 2nd left rib space at the sternal border = Pulmonic valve area
- 3rd left rib space at sternal border = Second pulmonic valve area
- 4th left rib space along sternal border = Tricuspid area
- 5th left rib space at the MCL = Mitral area
Heart sound that usually splits during inspiration
S2
S2 marks the initiation of this cardiac event
Diastole
S2 spliet is best heard in this auscultatory area
Pulmonic area
S1 is best heard at this location of the heart
The apex
S1 is the result from closure of these valves and indicates this cardiac event
AV valves close during systole
S2 is heard best over this area of the heart
The base
S3 is best evaluated at this location of the heart (Ventricular gallop)
Apex
S4 is best evaluated at this location of the heart (Atrial gallop)
Apex
Auscultatory area best for S1 split
Tricuspid
Events that might produce louder S1
- Increased in blood velocity (anemia, fever, hyperthyroidism, anxiety, exercise)
- Mitral stenosis
Events that might produce softer S1
- Complete heart block
- fibrillation
- increased overlying tissue, fat, or fluid
- Pulmonary hypertension
- Fibrosis or calcification of mitral valve preventing it from closing as forcibly
Events that might produce louder S2
- Systemic hypertension, syphilis of aortic valve, exitement, exercise
- Pulmonary hypertension, mitral stenosis, CHF
Events that might produce softer S2
- Arterial hypotension
- Aortic stenosis
- Pulmonic stenosis
- Overlying tissue, fat or fluid
Why does physiological splitting of S2 occur?
- Pressures are higher and depolarization occurs earlier on the left side of the heart
- Ejection times on the right are longer and the pulmonic valve closes later than the aortic vavle
RBBB splits S1 or S2?
Both
When splitting is unaffected by respiration
Fixed Splitting - Occurs with delatyed closure of the pulmonic vavle when output of the right ventricle is greater than that of the left
Splitting that occurs when closure of the aortic vavle is delyed so that the P2 occurs first followed by A2
Paradoxic (Reversed) Splitting - The interval between P2 and A1 is heard during expiration and disappears during inspiration
Reasons why S3 would be louder
-Increased ventricular filling
-Decreased compliance
-S3 is best hear when patient is in the left lateral recumbent
Words that most resemble rhythms in which S3 or S4 is heard
ken-TUCK-y = S3
TEN-nes-see = S4
Ejection clicks are made by
Semilunar Valves
Mid-to-late nonejection systolic clicks are made by
Mitral valve
The pulmoniary ejection click is best heard on ____ and is seldom heard on ____
Expiration, Inspiration
Pericardial friction rub may be heard widely but is more distinct toward the
Apex
Origin of murmur:
Inspiration increases murmur
Expiration decreases murmur
Right-sided chambers
Origin of murmur:
Vasalva and squatting to standing (rapidly for 30 secs) increases murmur
Standing to squatting rapidly and passive leg elevation to 45 degs (supine) decreases murmur
Hypertrophic cardiomyopathy
Origin of murmur:
Handgrip increases murmur
Mitral Regurgitation
Origin of murmur:
Transient arterial occulsion (brachial artery) increases murmur
Inhalation of amyl nitrate decreases murmur
Ventricular septal defect
Describe maneuvers that can distinguish aortic stenosis
No maneuver distinguishes this murmur; the diagnosis can be made by exclusion
Age and expected heart rates:
Newborn, 1 year, 3 years, 6 years and 10 years
Newborn
1 year: 120-170 bpm
3 years: 80-160 bpm
6 years: 75-115 bpm
10 years: 70-110 bpm
This accounts for most acquired murmurs in children
Rheumatic Fever
This murmur occurs in active healthy children between the ages of 3 to 7 yrs; often described as musical
A Still murmur
Systolic ejection murmurs may be heard over the pulmonic area in 90% of pregnant women, Murmur should not be louder than grade ___?
Grade II (Quiet but clearly audible)
How is mitral stenosis detected?
Heard with bell at the apex, with patient in left lateral decubitus position
Where can you best hear aortic stenosis? (you hear a crescendo-decrescendo sound) along the left sternal border and to carotid with palpable thrill)
Heard over aortic area; ejection sound at second right intercostal border; the more severe the stenosis, the later the peak of the murmur in systole
Where would you detect a subaortic stenosis?
Heard at apex and along left sternal border
Where is pumonic stenosis heard?
Over pulmonic area; it radiates to left and into neck; thrill in 2nd and 3rd left rib space
Triscuspid is best heard over this area
Heard with bell over tricuspid area
Mitral regurgitation is best heard here
Heard best at apex; loudes there, transmitted into left axilla
Where can you detect a mitral valve prolapse and describe the sound?
Heard at apex and left lower sternal border when patient is upright; late systolic murmur preceded by midsystolic clicks
How would you listen for an aortic regrugitation murmur?
Heard with diaphragm, patient sitting and leaning forward; ejection click heard in 2nd intercostal space; Austin-flint murmur heard with bell
Low-pitched, rumbling murmur at apex common in aortic regurgitation
Austin-Flint murmur
How would you listen for a tricspid regurge murmur?
Heard at left lower sternum, occasionally radiating a few centimeters to left
Physical finding:
Displacement of the apical impulse can be well lateral to MCL and downward; PMI is >2 cm
Left Ventricular Hypertrophy
Physical finding:
Lifts along the left sternal border in the 3rd and 4th rib spaces; occasional systolic retraction at the apex
Right Ventricular hypertrophy
Findings:
Triphasic friction rub (ventricular systole, early diastolic ventricular filling and late diastolic atrial systole); 90% of patients with this disease present with these findings
Pericarditis
Presentation:
Edema, ascites, dyspnea; Heart sounds are muffled, bp drops and pulse weakened and rapid and paradoxic pulse becomses exaggerated
Cardiac Tamponade
Common causes of tamponade
Pericarditis, aortic dissection and trauma
Enlargement of the right ventricle secondary to pulmonary malfunction
Cor Pulmonale
Desirable level of total cholesterol
200 mg/dL
Desirable level of LDL
100 mg/dL
Cardiac defects of the tetrology of Fallot
Ventricular Septal Defect
Pulmonary Stenosis
Dextroposistion of the aorta (overriding aorta)
Right Ventricular Hypertrophy
Clinical presentation of tetrology of Fallot
Paroxysmal dyspnea with loss of consciousness
Central cyanosis
Parasternal heave and precordial prominence
Single S2 is heard
Systolic ejection murmur is heard over the 3rd rib space
Holosystolic Murmur
Murmur is loud, coarse, high-pitched and best heard along the left sternal border in the 3rd-5th rib spaces
Ventricular Septal Defect
Neck vessels dilated and pulsate and the pulse pressure is wide
A harsh, loud, continuous murmur is often heard at the 1st-3rd rib spaces and the lower sternal border
Murmur is machine-like quality and usually does not change with posture
Patent Ductus Arteriosis
Causes a systolic ejection murmur that is diamond shaped, often loud, high pitch and harsh
Best heard over the pulmonic area
S2 split may be fairly wide
Atrial Septal defect
Major Manifestations of rheumatic fever according to Jones Criteria
Carditis
Polyarthritis
Chorea
Erythema marginatum
Subcutaneous nodule
Usually silent and painless until it produces sudden heart failure, strok or dysrhythmias
Mitral insufficiency