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26 Cards in this Set

  • Front
  • Back

Primary HA

No organic cause.


Migraines, tension, cluster.


50% are migraines.

Migraine s/s VS Cluster HA s/s

Migraine:


may have aura


associated with N/V



Cluster:


no aura


<1hr each waxing and waning


strictly unilateral


periorbital-temporal locations


Tension HA

dull/non-pulsating


bilateral, back to front


no photophobia


Temporal Arteritis

Inflammatory disease of cephalic arteries.



Blood supply to optic nerve so can lead to blindness.



Sed Rate may be Very High

Neuro Problems more associated with bleeds (2)

N/V



Photophobia

Ventriculo-Peritoneal Shunts

Placed to relieve incr ICP due to hydrocephalus.


Excess CSF shunted from ventricle to peritoneal cavity.



Complications: infection and malfunction



s/s : fever, distended/tender abdomen(from infx), bulging fontanels(infants) (from malfx shunt), change in LOC (malfx shunt)

Seizures : causes

Electrolyte imbalances (Ph changes)


Metabolic changes (fever, blood sugar imbalance)


Stress. Fatigue.


Nerve structure imbalance(hypoxia), tumors, trauma)


Medication noncompliance


ETOH/Benzo withdrawals

Status Epilepticus

Series of seizures without recovery period.



Emergency d/t risk for hypoxia.



No paralytics, still seizing in brain, only stopped external signs.

CPP : formula, normal limits

CPP = MAP - ICP



Normal = 70-100mm Hg

How to Manage increasing ICP

Increase the MAP : fluids, blood, pressors



Decrease the intra-cranial volume : diuretics, promote outflow, elevate HOB



Decrease vasodilation (which increases volume)

Cushings Response what it indicates, (5) signs

Indicates impending herniation



Hypertension : trying to perfuse brain tissue


Bradycardia : parasympathetic stimulation of vagus nerve.


Irregular Respirations


Widening Pulse Pressure


Pupillary Changes

Sub-arachnoid Hemorrhage/Hematoma

Skull->Dural->Arachnoid->cerebrum and pia



Common w contusions. Bleeding will contaminate CSF.



complications: seizures, incr ICP, hydrocephalus

Sub-Dural Hemorrhage/Hematoma

Skull->Dural->Arachnoid->cerebrum and pia



More likely venous and gradual bleeding.



High risk : elderly, anticoagulants, ETOH

Epidural Hemorrhage/Hematoma

Skull->Dural->Arachnoid->cerebrum and pia



Often d/t middle meningeal artery, r/t temporal skull fx



Arterial, rapidly expanding.



Fast progression to death. "talk and die" phenomenon of "lucid interval"

Spinal Cord Injuries : Complete

Transection.



No sensory/motor fx


Spinal Cord Injuries : Incomplete : Central Cord Syndrome

sensory/motor sx more pronounced in upper extremities

Spinal Cord Injuries : Incomplete : Anterior Cord Syndrome

loss of motor, pain and temp. sensation.


Preservation of vibration, touch and position perception.

Brown-Sequard Syndrome

Injury to one side of spine(hemisection)



Same side : paralysis/paresis, loss of pressure, touch vibration.



Other side : loss of pain and temp sensation.



(abnormalities on both sides of body, different abnormalities on both sides)

Innervation of Phrenic nerve and effects

Exits at C-3-4-5. C4="breath no more"



intercostal nerves for accessory muscle breathing exit at T1-T12. controls deep breathing, coughing.

Spinal Shock

problem with nerve TRANSMISSION.



No neuro fx below level of injury


No spinal reflexes, autonomic fx.


Flaccid paralysis.



Reflexes may return in weeks "stunned"


Neurogenic Shock

Problem with response to CATECHOLAMINES.



No sympathetic fx below injury.


vasodilation, hypotension, bradycardia


unopposed vagal(parasympathetic) influence



**skin is warm and dry below injury



Amyotrophic Lateral Sclerosis (Lou Gehrigs disease)

Neuro degenerative disease. Affects motor neurons(voluntary movment).



Cognitive fx usually spared, even when on ventilator, they are awake and alert.



frequent tripping, stumbling falls.

Mutiple Sclerosis

Autoimmune condition. CNS is attacked, demyelination.



Physical And cognitive disabilities(ALS-lou gehrigs is ONLY physical)

Myasthenia Gravis

Autoimmune disorder. Antibodies block Acetylcholine receptors. Causing fluctuating muscle weakness and fatiguability.



s/s : muscle weakness during activity, improving with rest.



Tensilon test used to help dx.



Tx : cholinesterase inhibitors, prednisone, plasmapherisis

Parkinsons Disease

Degenerative disease of CNS.



"TRAP" assessment



Tremors: at rest


Rigidity : cogwheel movement


Akinesia : slowness of movement


Postural Instability : balance problem



Brudzinskis Sign

Pt supine.



Passively flexes neck chin to chest



Pt involuntarily lifts leg = Positive