Cccla Soft Tissue Exam I

Front 1

TrP that causes a clinical pain complaint

Back 1

1) Active:

Front 2

TrP in one ms. that occurs concurrently with a TrP in another ms. One may have induced the other or both may stem from the same mechanical or neurological origin

Back 2

2) Associated:

Front 3

TrP at musculotendinous junction and/or at osseous attachment of the muscle that identifies the enthesopathy caused by unrelieved tension characteristic of the taut band that is produced by a central TrP

Back 3

3) Attachment:

Front 4

TrP closely associated with dysfunctional endplates & located near center of ms. belly

Back 4

4) Central:

Front 5

TrP responsible for activating one or more satellite TrPs. Clinically, a key TrP is identified when inactivation of that TrP also inactivates the satellite TrP

Back 5

5) Key:

Front 6

TrP that is clinically quiescent with respect to spontaneous pain. It is painful only when palpated. May have all the other clinical characteristics of an active TrP and always has a taut band that increases ms tension and restricts ROM

Back 6

6) Latent:

Front 7

Central TrP that was apparently activated directly by acute or chronic overload or repetitive overuse of the ms in which it occurs and was not activated as a result of TrP activity in another muscle

Back 7

7) Primary:

Front 8

A central TrP that was induced neurogenically or mechanically by the activity of a key TrP. May develop in the zone of reference of the key TrP, in an overloaded synergist that is substituting for “key” ms, in an antagonist countering increased tension of the “key” ms, or in a ms linked apparently only neurogenically to the key TrP.

Back 8

8) Satellite:

Front 9

The region of referred pain that is present in nearly every patient when the TrP is active

Back 9

1) Essential Pain Zone:

Front 10

General pain response of the patient, who winces, may cry out, and may withdraw in response to pressure applied on a TrP

Back 10

2) Jump Sign:

Front 11

:
Transient contraction of a group of tense muscle fibers (taut band) that traverse a TrP.
Contraction is in response to stimulation (usually by snapping palpation or needling) of the same TrP, or sometimes of a nearby TrP

Back 11

3) Local Twitch Response

Front 12

Soleplate ending where a terminal branch of the axon of a motor neuron makes synaptic contact with a striated muscle fiber

Back 12

4) Motor Endplate:

Front 13

Simultaneous expansion of the chest and contraction of abdominal muscles that pulls the abdomen inward during inhalation

Back 13

5) Paradoxical Respiration:

Front 14

Vasoconstriction (blanching), coldness, sweating, pilomotor response, ptosis, and/or hypersecretion that occur in a region separate from the TrP causing these phenomena. The phenomena usually appear in the same general area to which that TrP refers pain

Back 14

6) Referred Autonomic Phenomena:

Front 15

Region where some, but not all, patients experience referred pain beyond the essential pain zone, due to greater hyperirritability of a TrP

Back 15

7) Spillover Pain Zone:

Front 16

Tight Muscles
Hamstrings, Hip Flexors, Lumbar Erector Spinae

Weak Muscles
Gluteus Minimus, Medius, Maximus & AbdominalLayer

Back 16

Lower Crossed (Pelvic) Syndrome

Front 17

TightMuscles
Sternocleidomastoid, Levator Scapulae, Upper Trapezius, Pectoralis Major

Weak Muscles
Middle & Lower Trapezius, Rhomboids, Deep Neck Flexors

Back 17

Major Upper Crossed
(Proximal or Shoulder) Syndrome