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45 Cards in this Set
- Front
- Back
Pericardial Sac
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encases and protects the heart from trauma and infection
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Pericardial Space
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between parietal and visceral layers, 5-20 mL of fluid, cushions heart
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Atrioventricular Valves
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Tricuspid and Mitral, lie between atria and ventricles
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AV valves close
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at the beginning of ventricular contraction and prevent blood from flowing back into the atria from the ventricles
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AV valves open when
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the ventricle relaxes
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Semilunar valves
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aortic and pulmonic
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Semilunar valves prevent
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blood from flowing back into the ventricles during relaxation
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Semilunar valves open
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during ventricular contraction
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Semilunar valves close
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when the ventricles begin to relax
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SA node
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initiates each heartbeat
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SA node location
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junction of SVC and RA
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SA node generates electrical impulses
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60-100 times per minute
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AV node
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receives electrical impulses from the SA node
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AV node location
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lower part of atrial septum
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AV node generates electrical impulses
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40-60 times per minute
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Bundle of His
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branches into RBB and LBB
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R & L branches terminate into
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Purkinje fibers
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Bundle of His location
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interventricular septum
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Purkinje fibers
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spread wave of depolarization through ventricles
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Purkinje fibers pacemaker rate
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20-40 bpm
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RCA
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supplies blood to the right atrium and ventricle, inferior portion of LV, posterior septal wall, and the SA/AV nodes
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LMCA AKA
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Left main coronary artery
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LMCA
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has two major branches, the LAD and circumflex arteries
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LAD
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supplies blood to anterior wall of LV, anterior V septum, and apex of LV
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Circumflex Artery
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supplies blood to LA and lateral/posterior surfaces of LV
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S1
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AV valves close
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S2
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semilunar valves close
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S3
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occurs in CHF and valvular regurgitation
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S4
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occurs in atrial systole if resistance to ventricular filling is present
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Causes of S4
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cardiac hypertrophy, injury to ventricular wall
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Faster HR=
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decreased CO
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Sympathetic nerve fibers
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release norepinephrine increasing HR, atrial & ventricular contractility, & vasoconstriction
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Parasympathetic nerve fibers
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release neurotransmitter acetylcholine decreasing HR, atrial & ventricular contractility
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Baroreceptors
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specialized nerve endings affected by changes in arterial BP
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Increases in arterial pressure cause baroreceptors
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heart rate and arterial pressure decrease
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Decreases in arterial pressure cause baroreceptors to
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vasoconstrict and increase heart rate
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Stretch receptors
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respond to pressure changes that affect circulatory BV
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Decreased ADH(vasopressin) causes
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increased diuresis, decreased BV, decreased BP
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Increased ADH causes
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increase in BP and BV
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Renin
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potent vasoconstrictor
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Renin converts
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Angiotensin I to Angiotensin II
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Angiotensin II stimulates release of
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aldosterone
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Aldosterone
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promotes water and sodium retention causing increased BV and BP
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Capillaries
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exchange fluid and nutrients between blood and interstitial spaces
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Lymphatics
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drain tissues and return tissue fluid to blood
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