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158 Cards in this Set

  • Front
  • Back
gastro
stomach
necro
death
ocul/opthal/opt
eye
ped/o or pod
child/foot
phleb
vein
py
pus/fire
rhabdo
rodshaped/striated
thrombo
clot
tox
poison
neo
new
pachy
thick
pro
before
retro
behind
pexy
fixation or suspension
poiesis
formation of
py
pus/fire
rhabdo
rodshaped/striated
thrombo
clot
tox
poison
neo
new
pachy
thick
pro
before
retro
behind
pexy
fixation or suspension
poiesis
formation of
When infection gets all the way down to respiratory unit. Can be deadly. (occurs frequently in those with lungs already compromised)
pneumonia
30% of pneumonia cases.
PTs intervene largely here. Affects whole lobe of lung. if untreated, will spread to others.
bacterial pneumonia
patient PW: fever, shaking, chills, productive cough, dyspnea, CP if pleuritic (could just be in airspaces-no nerve endings) asymmetrical breathing pattern
bacterial pneumonia
Signs include:
BS: crackled, decreased, maybe wheezes
RR incr
VE incr
HR incr
temp incr
cyanosis (check nailbeds, mouth, eyes)
WBC high
ABGs:
PaO2 decr,
PaCo2 same (incr if too much of lung's involved)
pH (decr if PaCo2 incr)
CXR: can see where inf is-infiltrate
bacterial pneumonia
50 % of cases: patient PW:
fever
non-productive cough
dyspnea
RAPID ONSET
viral pneumonia
patient has following signs;
BS: crackles, may be decreased
RR incr
HR incr
WBCs same
ABGs:
PaO2 decr
PaCo2 same
CXR: can't determine if infiltrate's in alveoli or around
viral pneumonia
occurs in those who have trouble protecting their own airways. when object has surpassed branching of bronchiole tubes
aspirated pneumonia
patient PW:
BS: crackles, wheezes
RR incr
HR incr
cyanosis likely
WBC initially the same
ABGs:
paO2 decr
paCo2: same, unless it's really bad
pH: same " "
CXR: shows location of object, extreme inflammation, eaten-up lung tissue (looks worse than symptoms)
cough: not helpful in diagnosis
patient profile indicative in Dx
aspirated pneumonia
disease affects healthy individuals, then goes away.
pt c/o headache (predominant symptom), dry cough
mycoplasm (pneumonia)
tends to be epidemic. environment is moist. -waterborne
pt c/o dry cough, severe headache, WBC slight incr
legionella (pneumonia)
fungus causes pathology-hosts are typically those w/weak immune systems: AIDS, premature babies (no immune system from mother)
pneumocystic pneumonia (PCP)
patient PW:
low grade fever
progressive SOB
dry cough (heavy, hard to eject from deep in lungs)
weakness
weight loss
CXR: infiltrates present and visible
WBC no change
paCo2 usually incr
paO2: decr
pneumocystic pneumonia (PCP)

prevention is key for these people!
Prevention in those adults w/CD4 fell counts <200, clinical signs of opportunistic infection, and Hx of PCP:
supportive measures: O2, ventilation, positioning,
pharmacological Tx: 21 days
severe acute respiratory syndrome
started in china
SARS
patient PW:
high temp
dry cough
lymphopenia
leukopenia
CXR: borderline
SARS
postprandial
after a meal
exophthalamus
protrusion of eyeballs
myxedema
mucus edema caused by fluid retention
glycohemoglobin
molecule in hemoglobin that rises w/increased level of Blood sugar
populations at risk for TB:
elderly (weakened immune system)
alcohol dependent (immunity decr)
infants
prison inmates (no sunlight)
health care workers
patient c/o:
slight, non-productive cough
low grade fever
usually mid/lower lung involvement
lymph nodes
primary TB

though often none; is mild; asymptomatic.
positive skin test (manteaux) shows presence of antibodies, not active disease. drug ther. necessary to prevent post-primary
patient c/o:
fever
weight loss
cough
night sweats
CXR: often upper lobe involvement
pneumocele w/scarring around it
post-primary TB
symptom of TB: focal, calcified spot on chest radiograph (suggests granulomatous disease)
Ghon Lesion
Pt considerations for TB:
negative pressure isolation rm-2 weeks
cover mouth and nose when coughing
mask when leaving room for tests
PT considerations for TB
TB filtered masks
wash hands before and after
gloves for potential blood/body fluids
disinfect stethoscope b/w pts
annual manteaux
Tumor stage:
-tumor with no regional/distal mets
tumor stage I
-tumor w/hilar/peribronchial node involvement
tumor stage II
-(outside of lungs)locally advanced tumors w/mediastinal or cervical
lymph node mets & ext to chest wall, mediastinum, diaphragm
tumor stage III
tumors and distant mets
tumor stage IV
malignancy of epithelium of respiratory tract; pre-invasive lesions, small cell lung cancer (type I), non small lung cancer (type II)
bronchogenic carcinoma
malignancy in hilar region, linked to smoking, very rapid growth, very early metastasis - hilar and lung
small cell
most common type of lung cancer; almost 80% of lung cancers
non small cell
of nonsmall cell types:
-in periphery, tend to find these by accident. NOT LINKED TO SMOKING
adenocarcinomas
of nonsmall cell types:
usually more centrally-located (more dangerous due to central structures), hilar, LINKED TO SMOKING.slow metastasis, but central.
squamous
of nonsmall cell types:
-cells relatively undifferentiated-cell hasn't yet decided what it wants to be; peripheral, LINKED TO SMOKING. early metastasy.
large cell
of non small cells:
-least common
carcinoma
pt presents w/following:
weight loss
anorexia
fatigue
cough (doesn't go away for mos)
dyspnea
wheezing or decreased BS
sputum or not (if pneumonia-obstructed, no secretions)
hemoptysis (or not-coughing blood, only if tumor eats into capillary bed)
cancer!
pt pw:
airway compression/obstruction (wheezing and pneumonia w/partial obstruction, or pneumonia and volume loss w/full obstruction)
cough
hemoptysis
dyspnea
pain to chest, shoulder, back
weakness
fatigue
hoarseness from compression
atelectasis
difficulty swallowing
presence of central tumors
patient pw:
cough
sob
fatigue, weight loss
pleural pain if pleura involved
pleural effusion
peripheral tumors (are often undetected for some time)
-extreme apex of lung
-invade wall of chest rather than lung itself
(Lymphatics
Lower roots of the brachial plexus
Intercostal nerves
Stellate ganglion (Sympathetic nervous system)
Ribs
Vertebrae)
pancoast's tumor
pt pw:
-neuritic pain
-atrophy of arm and hand (reason for PT visit)
-Horner's syndrome (sinking of eye socket, ptosis of eyelid, constriction of the pupil, flushing of the side of the face
Pancoast's tumor
PT considerations for cancer
Bone metastasis-need to konw where they are, from whence they came, and what they can do.
Will have to deal with side effects of cancer tx (chemo, radiation, etc)
PT treatment for those w/cancer:
-aerobic exercise-will improve fatigue and weakness (side effects of chemo and radiation tx)
-prevention of contractures, skin breakdown, weakness
-pulmonary care to clear secretions as needed
platelet count is <50,000/uL
hemoglobin <10g/dL
WBC <3000/uL
absolute granulocytes <2500/uL

..can patient exercise?
no. low WBC especially-you can't risk giving them anything

(if hemoglobin is low, SaO2 is useless, bc it tells you that carrying sites are full, but these people don't have any hgB to carry it!)
Pulmonary condition, 'dead space'

blocking of bl vessel. no blood getting by. alveolar cap membrane affected. no o2 coming in, no Co2 coming off. person will appear hypoxemic. usually occurs pretty quickly (hypoxia and pain)
pulmonary emboli
Pulmonary condition:
pleura stay intact but lung tissue loses air. no air in lobe, but takes rest of lung and expands it, to fill in space that should have been taken up by affected lobe. everything is stretched and shifted over.
collapse!
pt PW:
BS: absent over site
tracheal shift toward site
collapse
pt pw:
temperature
decr saO2
pain in thorax-therefore not much air entry in area
--reinflating lungs will make normalize body temp
atelectasis
(collapse on a small scale)
pleural effusion
fluid collecting in CostoPhrenic angle (lowest portion of lung, where thorax meets gravity; most gravity-dependent part of lung..if fluid is present in pleura it will drop to this space).
Infected material residing in pleural space creating
BS
fever
increased WBC
SOB
empyema
pt PW:
BS
fever
increased WBC
SOB
TRACHEAL SHIFT away site
hemothorax/pneumothorax

(blood or air in pleural space)
With a tumor that is obstructing the left lower lobe bronchus, what will happen to the structures within the thorax?
to the left
With a right lower lobe pneumonia, what will happen to the structures within the thorax
Air will be replaced by an exudate. No change to the size or structure. SO, there will be no movement within the thorax.
With a right hemothorax, what will happen to the structures within the thorax?
Blood in the pleural space. This will shove the lung tissue away, shifting everything away from it..to the left.
-eucapnia
condition of normal Co2
scopy
to look into/view
surgical incision - cutting down center of sternum, easily accessible; heals well
midsternotomy
surgical incision: from T4-following fissure line around thru interspace; comes around to just before costochondral junction. ribs spread apart. (lung transplant incision; not ideal for heart surg)
posterior lateral thoracotomy
surgical incision-ribs spread apart. (pt will complain of aching at fulcrum points)
anterior lateral thoracotomy
surgical incision: cut underneath ribcage and "peeling it up"
clamshell incision for double lung transplant.
surgical procedure: (problem w/bronchi, common w/cancer): taking out piece of one, end to end anastamose.
bronchoplasty sleeve resection
surgical procedure: looking into lungs; only goes to segmental bronchus, after which you'll need a CT scan
bronchoscopy
surgical procedure: taking out lobe, leaving rest intact. (airway must come out with lung tissue) initially there'll be fluid where lobe was; shift away. when fluid reabsorbs it'll shift back
lobectomy
surgical procedure: a look into the mediastinum
mediastinoscopy
surgical procedure: binding together of pleura (when visceral and parietal pleura won't stay together).
pleurodesis
surg procedure: removal of whole lung. initially fills w/fluid; once this reabsorbs the other lung will have more space
pneumonectomy
surg procedure: removal of some air in lungs when there's too much that it's distended, impeding the healthy tissue from normal function. (volume reduction surgery)
pneumectomy
surg procedure: taking out segment of lung
segmental resection
surg procedure: removal of fluid from pleural space
thoracocentesis
surg procedure: noninvasive; going in with a stick to look at thorax
thorascopy/VATS
surgical procedure: hole in trachea, usually with hole for airway (now person can eat, move head, talk sometimes improve functionality)
tracheostomy
surgical procedure: removing lung segments without having to abide by anatomical boundaries
wedge resection
tests and measures following surgery:
vital signs
lab values
BS
cough ability
PT assessment for post operative care:
excursion
ROM of UE
strength
balance
functional ability
DNI/DNR
do not intubate; do not rescussitate
MVC
motor vehicle crash
IVDU
intravenous drug use
LVEF

..normal is?
left ventricle ejection fraction?

55%
TKA
total knee arthroplasty
NAD
no acute distress
PE
pulmonary embolis, pleural effusion, physical exam
childs larynx and vocal cords as compared to adult's
much higher
infant's
-tongue
-larynx
-epiglottis
large tongue,
high
high
angulation of infant's epiglottis
long, angled away from trachea
shape of infant's larynx

shape of adult's
funnel-shaped, narrowest at level of cricoid ring

cylindrical, straight down, narrowest at level of vocal cords
infant's airway vs adult's
less cartilaginous support in infant's; more easily compressed & occluded. small airways, less laminar flow, increased work of breathing
lungs of infant vs adult

compliance?
infants' lungs are less compliant, still dense, haven't completely inflated
thorax of infant as compared to adult
entirely cartilage (takes mos to ossify)
"back doors" between alveoli during obstruction

..present in..?
canals of lambert; pores of kahn.

not yet present in child; obstructions are thus more severe
lung tissue in infant vs adult:
from 0-6 years it grows nonlinearly

(during this time the incidence of lung disease is higher)
infant presents with:
tachypnea (>40 bpm)
cough - not powerful
cyanosis
pallor (lighter skin)
sweating (unusual in babies)
irritability
grunting (keeps airways open to exhale)
nasal flaring (babies are obligatory nose breathers)
retractions: breathing in so hard as to suck in intercostal spaces
drooling
respiratory distress
swollen larynx (obstructed airway) wheezes on inhale and exhale (sounds like a seal)
croup
First part of airway that can become inflamed. viral. must do trach if pt is obstructed
epiglottis
obstruction at level of bronchioles. no respiration can occur
bronchiolitis obliterans with organizing pneumonia (BOOP)

*respiration occurs at ends of bronchioles
common pathogen causing viral infection in lungs; very communicable Nov-March. carried by adults
RSV (respiratory syncytial virus)
pneumococcus
strep
H flu
not a flu; bacteria
infection deadly to kids; airways closed up. must be intubated.
B Pertussis (whooping cough)
lung immaturity; insufficient surfactant to keep alveoli open
Respiratory Distress Syndrome
treatment for respiratory distress syndrome:
ventilation support
positioning-optimize
secretion removal (if absolutely necessary)
early intervention
education
often sequelae of respiratory distress syndrome; iatrogenic due to high o2 and mechanical ventilatory pressure requirements
ABGs:
paO2 low
paCo2 high
BS: crackles, wheezes
bronchopulmonary dysplasia
pulmonary malformation whereby cilia are immobile
(line airways, move secretions in and out).
no beating= no clearing. if dysfunctional in lungs, probably elsewhere too
tracheoesophageal fistula:
no wall between esophagus and trachea. kid aspirates. surgical repair mandatory
meconium aspiration syndrome
combines pulmonary hypertension and obstructive airway disease with considerable inflammatory rxn. babies in distress during birth process: if meconium gets evacuated into amniotic fluid, it will enter baby's lungs. check amn fluid; if opaque = problematic (sticky, hard to clear from lungs)
diaphragmatic hernia
diaphragm never closed properly. colon protrudes through hole. child will turn blue upon first breath. won't know beforehand.
biggest risk factor for COPD
smoking
controlled drugs. drugs taken on regular basis
maintenance drugs
resp drugs used on intermittent basis to control symptoms (inhaled drug, used to prevent EIB for ex., shouldnt be used more often than a couple x/week. if needed more frequently, then it's not controlled.
rescue drugs
resp drugs for trauma: when breakthru symptoms can't be relieved. intravenous or intramuscular (when maintenance drugs and rescue drugs aren't working)
crisis drugs
routes of adm of resp drugs:

most direct?
oral
inhaled (most direct)
IV
cat of resp drugs:
mimic sympathetic system, bronchodilator, incr HR, BP, airway size, nervousness, insomnia
sympathomimetics

available as
-maintenance: long acting beta 2 agonist
-rescue: short-acting beta 2 agonist
-crisis
cat of resp drugs:
bronchodilator; acts by closing down parasympathetic nervous system. used for maintenance. not powerful enough for rescue or crisis
anticholinergic
cat of resp drugs:
bronchodilator (modest anti-inflammatory). many interactions. only crisis or maintenance. not inhaled.
methylxanthine
category of resp drug:
oral bronchodilator and anti-inflammatory- only used for maintenance, not rescue.
leukotriene modifier
category of resp drugs:
decreases airway edema and increases beta 2 receptor sensitivity. (steroids) appropriate for maintenance and crisis, not rescue. lots of side effects
anti-inflammatory drugs. corticosteroids.
category of resp drug that inhibits/stabilizes mast cells' provocation of vasoconstriction. only appropriate for maintenance. can take 4-6 weeks to start working. improves peak expiratory flow. to prevent EIB
mast cell stabilizers (cromones)
Seretide
Advair
Combivent

are..
combination drugs
Bacteriocidal
Bacteriostatic

are..
anti-microbial drugs
supplemental oxygen for:

maintenance:
rescue:
crisis:
maintenance: low flows
rescue: cannula or mask
crisis: 100% non breather mask (intubation)
pt PW:
pain (esp w/inspiration)
SOB
crepitus
asymmetrical breathing pattern
rib fx
treatment for rib fx:
pain control
breathing exercises
caution with manual techniques
2/more ribs fx (usually ribs 3-6). section of rib no longer connected to thorax. (negative pressure creates dyskinesia within rib)
flail chest
patient pw:
pain
sob
crepitus
CXR, BS
asymmetrical breathing pattern
paradoxical movement of certain section
flail chest
PT tx for small flail:

large flail:
small:
pain control
breathing ex
caution w/manual techniques

large:
may need splint
positive pressure ventilation (air forced in, entire thorax pushed out-until callous forms)
pt pw:
pain
sob
bs (can hear sucking sound)
cxr
nonfixed tracheal deviation

*first responder should..
pneumothorax


*place vaseline gauze to cover wound
difference b/w tension and non-tension pneumothorax:
tension pneumothorax: tearing of lung tissue with resulting tear in visceral pleura. air leaks into parietal pleural space (non-functional). common in tall male who's outgrowing his tissue extensibility
treatment for pneumothorax/hemothorax:
chest tube (rid thorax of substance)
breathing exercises
bl replacement
treat associated injuries
v/Q positioning
cardiac tamponade
person bleeding into pericardial sac
compresses heart.
heart can't accept blood..or function!!
myocardial contusion
heart ms is bruised; won't contract too strongly, but will be ok
aortic rupture
emergency
treatment for COPD:
mild
moderate
severe
very severe
mild: short-acting bronchodilator as needed
mod: regular rx w/long-acting beta 2 and rehab
sev: add inhaled steroid
very severe: add o2
treatment for GINA
very mild:
mild:
more severe:
severe:
very mild: short-acting bronchodilator as needed
mild: controlled rx, inhaled steroid
more severe: long-acting beta 2; possible leukotriene
severe: everything. oral steroids, methylxanthine, etc
treatmetn for CF:
(multi-system):
-pulmonary
-digestive
-pancreatic
-pulm: bronchodilators, mucolytics, anti-inflammatories
-dig: stool softeners, reflux
-pancreas:pancreatic enzymes, insulin