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130 Cards in this Set

  • Front
  • Back
  • 3rd side (hint)
P wave refers to
atrial depolarization
QRS complex
ventricular depolarization
ST segment
manifests MI, ischemia (problems with blood flow)
-elevated
-depressed
J point
beginning of ST segment. (junctional point, where deflection changes from vertical to horizontal)
EKG rhythm determined by
counting from one R to the next; looking for atrial and ventricular regularity
EKG rate determined by
count QRS complexes in a minute (or estimate).
if EKG rate is irregular?
count for a full minute!
measuring conduction from EKG: PR interval
time from when SA node fires, and when it gets through entire atria and fibrous band (beginning of P wave to beginning of ventricular depolarization)
how to calculate PR conduction?

normal values?
multiply baby boxes by .04

normal is 0.12-.2
measuring conduction at QRS:

normal values
beginning of Q to end of S (multiply by .04)

.04-.10
downward sloping ST segments
bad
upward sloping ST segment
not so bad
PVC characteristics (3)
-no P wave
-widened QRS complex
-compensatory pause
when is PVC significant?
there are more than 6/min., or they're multifocal (unidentical)
upside down T wave indicates?
ischemia, Mi (T wave flips weeks afterward)
cholesterol levels:
normal?
high?
120-200
>240
HDL desirable value?
at risk for CAD?
>60
<40
LDL desirable?
high?
<100
>160
triglycerides?
normal value
high value
<150
>200 mg/dL
predictive triglyceride levels in women?
-optimal?
-in men?
total: high
<5:1
High LDLs
homocysteine levels
>___ = increased risk for heart disease
what drops homocysteine?
>12
folic acid
C-reactive protein:
<___mg/L is considered low risk
>____mg/L =high risk of developing CAD
<1.0
>3.0
Anthropometrics/BMI
-underweight?
-normal?
overwt?
-obese?
<18.5
18.5-24.9
25-29.9
>30
minimum exercise parameters for at-risk for CAD individuals?

those with HTN?
aerobic, 20-30mins/day, 3-5x/week, intensity 70-85%max HR

calculate exact max HR
HTN can lead to? (3)
hypertrophied L ventricle
stroke (cerebral arteries)
coronary arteries (Heart trauma)
for diagnosis of HTN, must have ?
140/90
medical intervention for HTN (5)
ace inhibitors
diuretics
beta blockers
ca channel blockers
vasodilators
during systole:
great valves open, mitral and bicuspid valves close.
during diastole
AV valves open, great vessel valves close.
pathophysiology of valvular heart disease:
-infectious?
-congenital?
Rheumatic Fever (strep inf)

decreased leaflets,
mitral valve prolapse
symptoms:
SOB
syncope
dizziness
fatigue
slow onset over period of yrs
Left sided valvular heart disease
symptoms:
venous distention
tissue hypoxia without hypoxemia
syncope
dizziness
fatigue
slow onset of symptoms
Right sided valvular heart disease
signs:
forward failure
backward failure
murmur on heart sounds
normal EKG
valvular heart disease
Litotes
* Deliberate understatement of an idea
* By denying the contrary of the idea being affirmed
* Opposite of hyperbole
e.g. "War is not healthy for children and other living things.";
signs:
no crackles
adequate SaO2
cold extremities
decreased functional capacity
right sided heart failure
Grading murmurs:
1/6
2/6
3
4
5
1can only be heard w/careful listening
2-readily audible with stethoscope
3-a little louder than 2
4-as loud as 3, but with vibration attached
5-audible with edge of stethoscope
6-audible with naked ear
mitral stenosis:

heard as:
failure of mitral valve to open completely.

diastolic murmur
signs and symptoms:
SOB, CHF
orthopnea
PND
exercise intolerance
palpitations
syncope
dyspnea on exertion
peripheral edema w/R sided heart failure
mitral stenosis
a-fib (due to atrial enlargement)
systemic embolism
CHF
exercise intolerance
infection
may have R sided heart failure as well with peripheral edema
..complications of?
mitral valve stenosis or regurgitation
mitral regurgitation will be heard as?
murmur during systole
signs and symptoms:
SOB, CHF
orthopnea
PND
exercise intolerance
palpitations
light headedness, dizziness
syncope
DOE
peripheral edema w/right sided Hrt failure
mitral valve regurgitation
distinction b/w mitral stenosis and regurgitation?
listen to heart sounds! when in cardiac cycle the murmur occurs
signs and symptoms:
SOB, CHF
orthopnea
PND
ex intolerance
palpitations
light headedness
syncope
DOE
peripheral edema w/R sided heart failure
angina (due to LV hypertrophy)
aortic stenosis or regurgitation
dysrrhythmias, endocarditis, and LV hypertrophy are complications of..
aortic stenosis
L ventricle failure
pulmonary edema
endocarditis

..complications of?
aortic regurgitation
intervention for valvular disease?
-drug therapy (2)
anticoagulants
vasodilators (decrease afterload)
intervention for valvular heart disease (surgical (3))
balloon valvuloplasty (thru foramen ovale)
annuloplasty (repair-ring around valve)
valvular replacement
PT intervention for valvular heart disease
exercise (karvonen's for intensity; low end of range-long term sickness)
diet evaluation
education
exercise contraindicated in which valv. disorder?
aortic stenosis
symptoms:
skips a beat
SOB
headache
fatigue
dizziness
syncope
chest pain
dysrrhythmia
finding in EKG evidence can be done via:
at rest
Holtens (24/48 monitor)
King of hearts (pt presses button when symptoms come on)
during exercise
electrophysiologic studies are used when:
you think you can't catch dysrrhythmia on EKG and you think it might be lethal problem
dysrrhythmias : medical mgmt of:
pharmacology (2)
pacemakers(2)
other(2)
antiarrhythmics, beta blockers (decr HR)
pacemakers, Internal Cardiac defibrillator
Radiofrequency Catheter ablation (scars heart takes out defective loop)
revascularization
considerations for exercise prescription in pts with dysrrhythmias
Mode
frequency
duration
intensity (use karvonen no matter what-not to exceed rate of AICD)-stay at least 20 bpm below
PT intervention considerations for dysrrhythmic pts:
basic diet eval
stress reduction
education (about devices; if you feel arrhythmia coming on, sit down, call for help; don't drive)
common atrial arrhythmias:
atrial fibrillation
a-fib with slow ventricular response
a-fib with rapid response
can person w/atrial arrhythmia exercise?
yes, as long as it's not rapid, pt is on coumadin, and if BP holds
common arrhythmias: atrial waves are slower than a-fib, can actually see them, (p waves) (sawtooth appearance)
a flutter
uncommon arrhythmias: 4 or more beats consecutively:
v-tach
if person has atherosclerosis, he likely has (3)
Cerebral vascular disease
coronary artery disease
peripheral vascular diseae
angina occurs when
demand of heart exceeds supply of heart (can be blood volume or content issue)
angina exam: symptoms
have patient use words to describe
angina exam: occurrence
unstable vs stable (indicates predictability)
medical intervention for angina:
pharmacology :
nitro!
angina med intervention: revascularization route:
PTCA
arthrectomy
CABG
transmyocardial revascularization
PT intervention for angina:
exercise: mode, duration, frequency, intensity: karvonen's, keep below RPP threshold, symptoms: describe pain, nitr: have it with them; keep BP cuff with you!
karvonen's formula
(max HR-rest HR)x(40-85%) +HR rest
outside of ex, PT intervention for angina:
smoking cessation
diet eval
stress reduction
education
Ex prescription for angina:
20-30 mins, 3-5x/week, intensity: low end of karvonen
NYHA functional capacity w/angina/cardiac disease
I:
II:
III:
IV:
I: no limitation, often pt doesn't even know
II: slight limitation of physical activity, comfortable at rest
III: marked limitation of phys activity, comfortable at rest
IV: inability to carry on ay physical activity w/out discomfort. symptoms of heart failure /angina may present at rest, and increase on PE.
signs/symptoms:
pain/angina
SOB
nausea
light headedness
sense of doom
abnormally high or low HR and BP
Diaphoresis (cold, clammy skin)
MI
medical intervention (drug therapy for MI)
antianginals
decrease afterload
decreaes HR (-alols); beta blockers -obligatory for those who've infarcted; except asthma pts
increase contractility
lysing agents
anticoagulants
MI medical intervention:
PTCA (w/stent)
arthrectomy (shaving off plaque from arterial wall)
revascularization surgery (CABG, MIDCABG, offpump)
PT intervention for MI;
diet evaluation
stress reduction
pt education
exercise-freq, mode, duration, intensity-use karvonen's -must calculate exact HR, and RPE scale
During hospitalization, MI referrals: PT's role is: (5)
assess pt's hemodynamic response to activity
assess efficacy of medical mgmt
pt and family education
ascertain clinical data for prognosis/mgmt
identify/modify cardiac risk factors
examination procedure for MI
musculoskel/neuro screen
symptoms (angina pattern-know this before you move them at all)
Take vitals in supine, sit, stand, peak, and recovery
home situation
during hospitalization: levels of function:
1: bed positioning, PROM
2: bed to chair transfer
2+: bed to chair transfer, AAROM
3:warm up exercises, 50 ft amB
3+100 ft amB
4: 250 ft
5:500 ft, i flight stairs
during hospitalization: stop activity if:
-hypertension?
-hypotension?
-narrowing of pulse pressure to?
-EKG abnormalities?
-angina?
-fatigue?
-dyspnea?
>180/100
drop of 10-15mmHg
within 20 mmHG of each other
couplets, triplets, v-tach, a-fib, >10pvcs/min, change in rhythm
angina indicates need for medical mgmt
fatigue
dyspnea: record RR and SaO2
during hosp: pt is home ready when:
-RPE and HR
ex
ex progression
pt and famliy ed
OP CR
accurate self monitoring of RPE and HR
ex prescribed based on IP performance w/mode, intensity, freq, adn duration is written
ed completed
referral to OP CR given
cardiomyopathy defn:

EF<___?
poor contractility of all myocardial segments

<25%
5 causes of cardiomyopathy:
extensive multi-vessel CAD
viral
toxic
post-partum
congenital
2 types of cardiomyopathy:
ischemic, nonischemic
ischemic cardiomyopathy:
results from heart ms damage from CAD
nonischemic (3) cardiomyopathy:
dilated
hypertrophic
restrictive
dilated cardiomyopathy: chambers dilated, fibers pulled apart, lengthened, interspace too large for effective contraction:
vent volume__
ven mass__
systolic fcn__
diastolic fcn__
incr
decr
decr
stiffness incr, filling pressure incr
signs/symptoms:
fatigue
weakness
SOB
DOE
orthopnea
edema
wt gain
arrhythmias
systolic murmur S3
echo: englarged chamber size
cardiac cath
EKG: arrhythmias
dilated cardiomyopathy
treatment for cardiomyopathy (drug therapy (5))
digoxin
diuretic
vasodilator
beta blocker
calcium channel blocker
treatment for dilated cardiomyopathy (surg)
LVAD
pacemaker
AICD
ventriculectomy
transplant
hypertrophic cardiomyopathy
chamber size gets smaller, spetal wall blocks aorta, and blood can't get out. (diastole is ok, systole is problem)
hypertrophic cardiomyopathy:
-vent volume:
-vent mass:
-systolic fcn:
diastolic fcn:
decr
incr
EF normal, outflow obstruction
stiffness increased, filling pressure increased
signs/symptoms:
SOB
dizziness
fainting
angina
cardiac arrhythmias that can lead to sudden death
heart murmur can be heard
hypertrophic cardiomyopathy
hypertrophic cardiomyopathy intervention:
-alcohol ablation
-myectomy
-produces chem MI (subaortic) thins septum to enlarge outflow tract
-area of ms removed to increase chamber size
restrictive cardiomyopathy
-vent volume
-vent mass
-systolic fcn
-diastolic fcn
same
rigidity
EF decreases
stiffness increased, decreased vent filling, filling PRESSURE increased
Prior conditions of
cardiac ischemia/infarction
valvular dysfunction
pulmonary disease
cardiomyopathy
..can lead to?
chronic heart failure
symptoms/signs:
fatigue
cold extrems
SOB
decreased ability to perform ADLs
orthopnea
PND
mottling of skin
peripheral edema
PaO2 decreased
decreased fcnl capacit
crackles
cardiac wheezing
s3 maybe s4
chronic heart failure
chronic heart failure drug therapy: (5)
beta blockers
digoxin
vasodilators
diuretics
ca channel blockers
PT intervention in chronic heart failure:
exercise: intensity karvonen's, stress reduction, eduation, diet eval
revascularization surgeries for chronic heart failure:
CABG
balloon angioplasty (PTCA)
TMR
Revascularization: balloon angioplasty:
tip of catheter into groin thru fem A, adcanced to obstr, and inflated and released, increasing bl flow
Post surgery: quick PT assessment of pt:
vital signs with mobility
sensation
pain mgmt
bs
cough ability
functional assessment/early amb
active motion
main issues w/post-operative care:
pulmonary
mobility
teaching
home-ex program
valvular surgeries:
commissurotomy
balloon valvuloplasty
valvular repair
valvular replacement
surgeries for cardiac arrhythmias
ablation
AICD
surgical mgmt of heart failure (3)
LVAD
Jarvik (pump attached to L vent; pumps oxygenated bl throughout body)
ventriculectomy
surgical mgmt of hypertrophic cardiomyopathy:
ethynol ablation (takes off piece of septum)
pharmacology:
lipid reduction category:
antihyperlipidemics,
all end in -statin
adverse effects: stressed GI
for triglyceride reduction
lifestyle changes:
smoking cessation
control of Bl glucose
limit/cease alcohol consumption
risk factor mod for pts w/HTN
stop smoking
stop alcohol
exercise
maintain healthy bodyweight
healthy diet
manage stress
pharmacology for HTN:
ACE inhibitor (#1 antihypertensive)
diuretic
beta blockers
ca channel blockers
angiotensin
CAD pharmacology
HTN drugs (ace, diuretic, beta blockers, ca channel blockers, angiotensin)
nitro
anticoagulants
thrombolytics
cardiotonics (incr contractility)
intervention for valvular disorders:
-lifestyle:
-pharm:
surgical repair (no lifestyle)

diuretics
anticoagulants
antiarrhythmics (if atria is separating)
cardiotonics
antibiotics (vegetation?)
pharm for dysrrhythmias:
-lifestyle
-pharm
none
beta blockers, anticoagulants, ca channel blockers, antiarrhythmics (awful drug; last resort)
lifestyle mods for chronic heart failure and cardiomyopathy:
sodium
sat fat, cholesterol
limit alcohol
exercise
stop smoking
weigh daily (>3 lbs rapidly=fluid ret'n; call doc)
reduce stress
Risk factors for CAD (9)
HTN
smoking
cholesterol
family history
obesity
sedentary lifestyle
diabetes
gender
S1:
___:
S2:
___:
mitral and tricuspids close
systole
great vessel valves close
diastole
aortic heard best where?
at base of R
pulmonic heard best where?
at base of L
S3 sound:
abnormal; best heard w/light pressure over apex during early diastole (rapid filling); reflects decreased ventricular compliance or increased ventricular diastolic volume .
S3 heard with:
CHF, CAD, incomplete valves, VSD, PDA)
S4 sound:
occurs just before 1st heart sound (late diastolic filling, when atria contract). usually developed by those who've just ahd heart attack. causes: HTN, aortic stenosis, L vent MI
auscultation of lungs in assessing cardiac fcning?
important! crackles at bases? is cardiac system affecting the lungs?
C-reactive protein:
inflammation marker; used to predict new coronary events in pts w/unstable angina and acute MI. increased in indvs w/recent tissue injury, or generalized infection
CK:
abnormal:
marker of cell death (ms, heart, brain)
>200
CK-MB:
abnormal:
marker of myocardial cell death
>10
CK-MB index:
abnormal:
differentiating noncardiac and cardiac sources of elevated Ck-MB
>6
troponin:
no "normal" amt is present in blood. more immediate indicator than cks. indicates cell death in cardiac cycle.
.01-.1; >.1 is significant.
BNP
brain natriuretic peptide: used to diagnose heart failure. differentiate b/w heart and lungs. (may be low in pts w/ACE, beta blockers)
St elevation or depression and T wave inversion are signs of:
ischemia/infarction
significant Q waves:
long term marker of MI (chronic); don't look for these acutely
MIBI vs thallium
same effect; looking for perfusion, though MIBI is rid from system quicker.