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8 Cards in this Set

  • Front
  • Back
Characteristics of Creatine Kinase (MB)
dimer of M and B subunits
increased following MI
CK and CKMB abnormal withn 2-8 hrs of onset of symptoms, peak within 36 and normal within 2-3 days
only half of pt have increase in CKMB at time they come into hospital
characteristics of troponins
complex of three regulatory proteins bound to contractile apparatus of striated muscle
cardiac contains I and T
relationship of troponins to prognosis and clinical decisions with chest pain
increases are more specific than CKMB for MI
same timing with CKMB (hours after symptoms)
levels persist for 7-10 days, longer than CKMB
heart is only source of troponins
serum concentrations are increased in some pt who lack def EKG or CKMB evidence of AMI
not specifi for CA etiology of MI, increased by any cardiac injury
relation of cardiac natriuretic peptides to HF
ANP, BNP increase in CHF
ANP released in response to atrial distension
BNP realses in response to ventricular overload
CNP in response to endothelial stress
CKMB is not usualy increased in
angina, endocarditis, exercise testing, IM injection, PE
CKMB increases in
skeletal muscle (MD, trauma, hypothyroid, stroke, psych)
cardiac muscle (MI, trauma)
Smooth Musc (labor, delivery, intest infarct, shock)
which natriuretic peptise correlates with severe heart failure
BNP is used to dx what?
HF in ED pt
monitoring therapy of HF
prognosis of acute coronary syndromes