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26 Cards in this Set

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uses for digoxin
CHF (+inotropy/contractility)
atrial fibrillation (decrease conduction of AV node)
Side effect is decreased QT and prolonged PR and scooping of ST
digoxin
why think Van Gogh with Digoxin
increases parasympathetics-->blurry vision, nausea, diarrhea
antidote for digoxin
normalize K+, lidocaine
anti-dig FAB
increases the toxicity of digoxin
renal failure
hypokalemia
five classes of antiarrhythmic drugs
I sodium blockers
II B-blockers
III-K+ blockers
IV Ca2+ blockers
V others
block fast acting Na+ in phase 0 prolonging AP
Quinidine
Amiodarone
Procainamide
Disopyramide
class Ia
Queen Amy Proclaims Diso's Pyramide
SA is cinchonism-headache, tinnitus, thrombocytopenia, torsade de pointes
Quinidine
Torsade de pointes is from increase in QT
Class Ia with SA of reversible SLE-like effects
procainamide
used for ventricular arrhytmias (post MI) and digitalis induced arrhythmias
lidocaine,
mexiletine
tocainide
ClassIb
what does mexiletine do to AP
decrease
this is a class Ib drug
only used as a last resort for ventricular arrhythmias
Flecainide
encainide
propafenone
classIc
4 classes of drugs that act on AV node that prolong PR interval
-b-blockers
-digoxin (cardicac glycoside)
-calcium channel blockers
-cholinesterase inhibitors/drugs that increase cholinergic activity
-these drugs either increase refractory time/increase calcium/increase cholin. activity
why would you not use propranolol/beta blockers in diabetic
masks signs of hypoglycemia
side effect of metoprolol
dyslipidemia
K+ blockers
satalol
ibutilide
bretylium
amiodarone
increase AP, QT and refractory period
ClassIa (Quinidone, amiodarone, procainamide, disopyramide)
Class III (amiodarone, satalol, ibutilide, bretylium)
used when other antiarrhythmics fail
class III
ClassIc
safe to use in Wolff-Parkinson-White
Amiodarone
side effects of satalol
pulmonary fibrosis
hepatotoxicity
hypo/hyperthyroidism
ca2+ channel blockers used in heart
verapamil
diltiazam
3 indications for diltiezem (Ca2+ channel blocker)
1)coronary artery dilation--> angina/variant angina
2)AV nodal blocker--> SVT/rapid rate afib/flutter
3)vasodilator-->hypertension
side effects of Ca 2+ channel blockers
flushing, edema, AV block, torsades de pointes
drug of choice for diagnosing/abolishing AV nodal arrhythmias
adenosine
depresses ectopic pacemaker in digoxin toxicity
K+
effective in torsades de pointes and digoxin toxicity
Mg2+