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13 Cards in this Set

  • Front
  • Back
symptoms
angina - all types including microvascular
dyspnea
CHF
Physical assessment
S4, MR/TR murmur, T wave inversion for ischemia, peaked T wave for acute MI, ventricle arrythmias, conduction defects
end stage ischemia disease secondary effects
LV/RV systolic dysfunction
CHF
LV thrombus
Ischemia Changes Cascade
perfusion defect, diastolic function abnormality, segmental wall motion abnormality, ecg changes, chest pain, myocaridal infarction.
LV diastolic dysfunction
impaired relaxation, reduction in early diastolic filling, reduced compliance, increased end diastolic presssure
segmental wall motion abnormalities
normal - increase in thickness for systolic
ischemic - not thicken, may be thin
Wall motion scoring - normal and hypokinetic
Normal - endocardial motion and wall thickening normal
Hypokinetic - reduced endocardial motion and wall thickening during systole, <40%
Wall motion scoring: akinetic, dyskinetic and aneurysmal
Akinetic - absence of inward endocardial motion or thickening, <10%
Dyskinetic - outward motion or bulging of endocardium during systole
Aneurusmal - fixed defect during systole and diastole
Wall motion scoring
normal 1
hypo 2
akinetic 3
dyskinetic 4
aneurysm 5
coronary supply
LAD - anterior wall and septum, LV apex
Circumflex - lateral wall, posterior wall
RCA - RV apex, RV free wall, inferior wall, inferior septum
Myocardial ischemia
complete blockage occurs for
<4 hours ischemic
>4 hrs infarcted necrosis, irreversible
Ischemia - stunning and hibernating
Stunning - viable myocardium salvaged by reperfusion, recovery may not be immediate
Hibernating - ischemic myocardium supplied by narrow coronary, prolonged post ischemia dysfunction
Ventricular remodeling
after cokmplete occlusion and infarction, necrotic myocardium becomes fibrous and scarred. thinner and dense. may cause ventricular arrhythmias, CHF, diastolic dysfunction