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13 Cards in this Set
- Front
- Back
symptoms
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angina - all types including microvascular
dyspnea CHF |
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Physical assessment
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S4, MR/TR murmur, T wave inversion for ischemia, peaked T wave for acute MI, ventricle arrythmias, conduction defects
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end stage ischemia disease secondary effects
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LV/RV systolic dysfunction
CHF LV thrombus |
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Ischemia Changes Cascade
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perfusion defect, diastolic function abnormality, segmental wall motion abnormality, ecg changes, chest pain, myocaridal infarction.
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LV diastolic dysfunction
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impaired relaxation, reduction in early diastolic filling, reduced compliance, increased end diastolic presssure
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segmental wall motion abnormalities
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normal - increase in thickness for systolic
ischemic - not thicken, may be thin |
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Wall motion scoring - normal and hypokinetic
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Normal - endocardial motion and wall thickening normal
Hypokinetic - reduced endocardial motion and wall thickening during systole, <40% |
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Wall motion scoring: akinetic, dyskinetic and aneurysmal
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Akinetic - absence of inward endocardial motion or thickening, <10%
Dyskinetic - outward motion or bulging of endocardium during systole Aneurusmal - fixed defect during systole and diastole |
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Wall motion scoring
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normal 1
hypo 2 akinetic 3 dyskinetic 4 aneurysm 5 |
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coronary supply
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LAD - anterior wall and septum, LV apex
Circumflex - lateral wall, posterior wall RCA - RV apex, RV free wall, inferior wall, inferior septum |
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Myocardial ischemia
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complete blockage occurs for
<4 hours ischemic >4 hrs infarcted necrosis, irreversible |
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Ischemia - stunning and hibernating
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Stunning - viable myocardium salvaged by reperfusion, recovery may not be immediate
Hibernating - ischemic myocardium supplied by narrow coronary, prolonged post ischemia dysfunction |
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Ventricular remodeling
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after cokmplete occlusion and infarction, necrotic myocardium becomes fibrous and scarred. thinner and dense. may cause ventricular arrhythmias, CHF, diastolic dysfunction
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