• Shuffle
    Toggle On
    Toggle Off
  • Alphabetize
    Toggle On
    Toggle Off
  • Front First
    Toggle On
    Toggle Off
  • Both Sides
    Toggle On
    Toggle Off
  • Read
    Toggle On
    Toggle Off
Reading...
Front

Card Range To Study

through

image

Play button

image

Play button

image

Progress

1/79

Click to flip

Use LEFT and RIGHT arrow keys to navigate between flashcards;

Use UP and DOWN arrow keys to flip the card;

H to show hint;

A reads text to speech;

79 Cards in this Set

  • Front
  • Back
what do ureters pass under?
uterine arty and ductus deferens
what creates the negative charge barier in basement membrane
heparan sulfate
typical osmotic pressure in bowman's space
0
relationship between RBF and hematocrite
RBF = RPF/(1-Hct)
relationship between ERPF and RPF
ERPF underestimates RPF by 10%
effect of NE on afferet/efferent
constricts afferent and efferent
effect of prostaglandins on afferent/efferent
dilates afferent
effect of sympathetics on afferent/efferent
constricts both
effect of ADH on afferentefferent
constricts Efferent (100x more than NE)
effect of AII on Afferent/Efferent
constricts Efferent
effect of AN on Afferent/efferent
dilates afferent
effect of NSAIDS on afferent/efferent
Constricts afferent
effect on RPF, GFR and FF of dilating afferent
increase RPF
Increase GFR
FF stays the same
effect on RPF, GFR and FF of constricting efferent
decrease RPF
increase GFR
increase FF
effects of ACEi on afferent/efferent
dilates afferent because of inhibition of AII
what happens to RFP, GFR, FF when plasma protein increases
RFP unchanged
GFR down
FFdown
effect on RPF, GFR and FF of constriction of ureter
RPF unchanged
GFR down
FF down
with ADH present, what is the free water clearance
<0
what is free water clearance in isotonic urine
0
by what point in nephron is glucose fully reabsorbed
proximal tubule
what is threshold for glucosuria
200 mb
where does active transport for amino acids happen
proximal tubulew
where in the nephron does PTH work
distal convuluted tubule, where it controls reabsorption ofCa++
where is NaH exchange in the kidney
proximal tubule
where is NaCl cotransport in the kidney
distal convoluted tubule
is NaK ATPase lumenal or basolateral
on blood side
where is there Na-Ca++ exchange in the kdney
on blood side in distal convoluted tubule so that Ca can be reabsorbed (which is under control of PTH)
Where is NKCC pump
lumenal in Thick ascending limb
where do Mg and Ca get reabsorbed
via sneaking around cells of teh TAL
where is urine made hypertonic
thin descending loop
what is threshold for glucosuria
200 mb
where does active transport for amino acids happen
proximal tubulew
where in the nephron does PTH work
distal convuluted tubule, where it controls reabsorption ofCa++
where is NaH exchange in the kidney
proximal tubule
where is NaCl cotransport in the kidney
distal convoluted tubule
is NaK ATPase lumenal or basolateral
on blood side
where is there Na-Ca++ exchange in the kdney
on blood side in distal convoluted tubule so that Ca can be reabsorbed (which is under control of PTH)
Where is NKCC pump
lumenal in Thick ascending limb
where do Mg and Ca get reabsorbed
via sneaking around cells of teh TAL
where is urine made hypertonic
thin descending loop
where is urine made hypotonic (i.e., which is the diluting segment)
early distal convoluted tubule
what do intercalated cells have
KH ATPase , lumenally (only place)
bicarb-CL exchange, on blood side (only place)
which part of kidney is impermeable to water
thick ascending loop
which is the diluting segment
TAL and early DCT
what receptor does ADH work on in collecting tubule
V2
what cells are aquaporins in
principal cells of collecting tubules
where does renin act on angiotensinogen
liver
where does ACE work on angiotensin I
in lung capillaries
what provides a check on the RAS
ANP
what secretes renin
juxtablomerular apparatus
what stimulates renin release
decreased renal arterial pressureincreased sympathetic discharge (via Beta-1 receptors
how can NSAIDS cause acute renal failure
by inhibiting the prostaglandins that would otherwise keep the afferent areterioles vasodilated, which the kidney is in a high vasoconstrictive state
decreased pH decreased PCO2, decreased bicarb
metabolic acidosis
pH up
PCO2 up
bicarb up
metabolic alkalosis
pH down
PCO2 up
bicarb up
respiratory acidosis
pH up
PCO2 down
bicarb down
respiratory alkalosis
causes of respiratory alkalosis
hyperventilation
aspirin ingestion
caues of respiratory acidosis
ahypoventilation due to:
- airway obstruction
- acute lung disease
- chronic lung disease
- opioids, narcotics, sedatives
- weakened respiratory muscles
Type 1 renal tubular acidosis
defect in H+ pump and failure to acidify urine
Type 2 renal tubular acidosis
renal loss of biarb
type 4 renal tubular acidosis
hypoaldosteronism, leading to hypokalemia, leading to inhibition of ammonia excretion
where is ammonia secreted
early proximal tubule (I think in exchange for Chlorine)
bilateral renal agenesis
Potters syndrome
Nephritic or Nephrotic: Acute poststerptotococcal glomerulonephritis
nephritic
Nephritic or Nephrotic: Membranoproliferative
nephritic
Nephritic or Nephrotic: RPGN
Nephritic
Nephritic or Nephrotic: Goodpastures
Nephritic
Nephritic or Nephrotic: IgA Nephropathy
Nephritic
Nephritic or Nephrotic: Membranous Glomerulonephritis
nephrotic
Nephritic or Nephrotic: FSGS
nephrotic
Nephritic or Nephrotic: Diabetic nephropathy
nephrotic
Nephritic or Nephrotic: amyloidosis
nephrotic
mesangial deposits
IgA nephropathy
large irregular subepithelial deposits or humps
acute glomerulonephritis
which kind of kidney stones are worsened by alkaluria? improved?
Struvites are made worse,
Cystine are made better
acute generalitzed infarction of cortices of both kidneys
diffuse cortical necrosis
due to combination of vasospasm adn DIC
associated with obstetric catastrophes and septic shodk
typical cause of prerenal azotemia
hypotension

(and so high urine osmolality because water conserved)
part of nephron that gets Fanconi's syndrome
proximal tubule transport
cortical and medullary cysts
long standing dialysis