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111 Cards in this Set

  • Front
  • Back
Hormones –
chemicals produced by endocrine (ductless) glands
Are released by glands and circulate through bloodstream, taking their effect by attaching to receptors on organs or other glands
hormones
WHAT DOES IT MEAN TO ORGANIZE THE SEX HORMONES
ITS DONE AT SENSITIVE STAGE OF DEVELOPMENT SHORTLY BEFORE THE BIRTH IN HUMANS AND DETERMINE WHETHER THE BRAIN AND BODY WILL DEVELOP MALE OR FEMALE CHARACTERISTICS
WHEN CAN ACTIVATING EFFECTS HAPPEN
ANY TIME IN LIFE AND ACTAVATES ITS EFFECTS ON A N ORGAN

DURING PUBERTY AND HORMONES CAN INDUCE LONG LASTING STRUCTURAL CHANGES
WHAT HAPPENS TO THE HORMONES IN EARLY IN LIFE?
ORGANIZE BODY DEVELOPMENT
WHAT ARE THE WAYS HORMONES INFLUENCE THE DEVELOPMENT OF
OF SEX
1.. INFLUENCE THE DEVELOPMENT FROM CONCEPTION TO SEXUAL MATURITY OF ANATOMICAL, PHYSIOLOGICAL AND AND BEHAVIORAL CHARACTERISTICS DISTINGUISH FROM MALE AND FEMALE

2. IT ACTIVATES THE HORMONES TO THE REPRODUCTION RELATED BEHAVIOR OF SEXUALLY MATURE ADULTS
WHAT HORMONES INFLUENCE SEXUAL DEVELOPMENT AND THE ACTIVATION OF ADULT SEXUAL BEHAVIOR
STEROID HORMONES
WHICH ARE SYNTHESIZED FROM CHOLESTEROL A TYPE OF FAT MOLECULE
WHAT DO THE GONADS DO
CREATE SPERM AND EGG CELLS
PRODUCE AND RELEASE STEROID HORMONES

BOTH TESTES AND OVARIES RELEASE THE SAME HORMONES
TWO MAIN CLASSES OF HORMONES
ANDROGEN AND ESTROGEN
GONADS ARE
MALE TESTES
FEMALE OVARIES
MOST COMMON ANDROGEN
TESTOSTERONE
MOST COMMON ESTROGEN'S
ESTRADIOL
OVARIES RELEASE WHAT
MORE ESTROGEN THAN ANDROGEN
TESTES RELEASE WHAT
MORE ANDROGEN THAN ESTROGEN'S
WHAT DO WE NEED TO AVOID REGARDING ANDROGEN'S AND ESTROGEN'S
NAMING THEM THE MALE AND FEMALE SEX HORMONES BECAUSE THEY DON'T
THE THIRD CLASS OF STEROID HORMONES RELEASED BY THE OVARIES AND TESTS ARE CALLED`
PROGESTINS
THE MOST COMMON PROGESTINS IS
PROGESTERONE
FUNCTION OF PROGESTERONE
FEMALES PREPARES THE UTERUS AND BREASTS FOR PG

MALE FUNCTION IS UNKNOWN
ADRENAL CORTEX
LAYER ON ADRENAL GLANDS REGULATES GLUCOSE AND NA LEVELS IN BLOOD (NOT THOUGHT OF AS A SEX GLAND IT DOES RELEASE SMALL AMTS OF SEX STEROIDS THAT ARE RELEASED BY THE GONADS
WHAT ARE THE FACTORS THAT HELP INFLUENCE THE HORMONES IN SEXUAL DEVELOPMENT
CHROMOSOMES

DEVELOPMENT OF INTERNAL REPRODUCTIVE SYSTEM

DEVELOPMENT OF EXTERNAL GENITALIA

PUBERTY
AT WHAT STAGE IS THE DEVELOPMENT OF INTERNAL REPRODUCTIVE SYSTEM
6 WEEK GESTATION
WHAT ABOUT EMBRYOS ARE
unisex until this differentiation – equally cable of forming male and female pattern REGARDLESS OF CHROMOSOMES
prompt development of male pattern, no androgens result in female pattern
ANDROGENS
Development of external genitalia
3 MONTHS INTO GESTATION

Again, androgens are important and differentiate the male from female pattern
AA EMBRYO AT ONE MONTH IS SPONTANEOUS ABORTED HOW CAN WE TELL IF IT WAS MALE OR FEMALE
ONLY BY CHECKING THE CHROMOSOMES THE BODY STRUCTURE LOOKS THE SAME
WHAT DOES IT MEAN TO BE DIMORPHIC
COMES IN TWO STANDARD MODELS FEMALE AND MALE
AT 6 WEEKS OF CONCEPTION WHY IS THE XX AND XY INDIVIDUALS IDENTICAL?
BECAUSE EACH HAS THE SAME PAIR OF GONADAL STRUCTURES CALLED THE PRIMORDIAL GONADS
WHAT IS THE PRIMORDIAL GONADS ARE?
PRIMORDIAL MEANS EXISTING AT THE BEGINNING

AND CONTAINS A CORTEX AND CONTAINS A MEDULLA
WHEN DOES SEXUAL DIFFERENTIATION BEGIN
AT FERTILIZATION WITH THE PRODUCTION OF ONE OF TWO KINDS OF ZYGOTES

(XX) FEMALE (XY) MALE
WHAT NORMALLY DETERMINES IF THE EMBRYO WILL BE MALE OR FEMALE
BE CAUTIOUS

GENETIC INFORMATION ON THE SEX CHROMOSOMES
CORTEX ON THE PRIMORDIAL GONADS
OUTER COVERING
WHICH COULD DEVELOP INTO OVARY
MEDULLA ON THE PRIMORDIAL GONADS
IS AN INTERNAL CORE WHICH COULD DEVELOP INTO TESTIS
WHEN DOES SEXUAL DIFFERENTIATION BEGINS
AT 6 WEEKS PRENATAL OR POST CONCEPTION
WHAT CAUSES THE MEDULLA OF EACH PRIMORDIAL GONAD TO GROW AND DEVELOP INTO A TESTIS
AT 6 WEEKS AFTER CONCEPTION, THE SRY GENE ON THE Y CHROMOSOME OF THE MALE TRIGGERS TE SYTHESIS OF SRY PROTEIN AND THIS PROTEIN CAUSES THE MEDULLA TO GROW AND DEVELOP INTO TESTES
WHAT CAUSES THE CORTEX OF THE PRIMORDIAL GONAD TO DEVELOP INTO AN OVARY
THERE IS NO Y CHROMOSOME PRESENT AND NO FEMALE COUNTERPART OF SRY PROTEIN
HOW ARE THE DEVELOPMENT OF THE INTERNAL DUCTS MALE AND FEMALE REPRODUCTIVE SYSTEMS FROM WHAT?
WOLFFIAN --------MALE
MULLERIAN ---- FEMALE
AT THE SIX WEEKS POST CONCEPTION EACH FETUS HAS?
THE SAME PAIR OF GONADAL STRUCTURES THE MEDULLA AND CORTEX
WHAT TRIGGERS THE DEVELOPMENT OF PRIMORDIAL GONWAD MEDULLA INTO TESTIS
THE Y CHROMOSOME SRY GENE
WHAT DOES THE SRY GENE MAKE
THE SRY PROTEIN THAT TRIGGERS THE MEDULLA INTO TESTIS
iF NO SRY GENE THEN WHAT HAPPENS
OVARIES DEVELOP
WHAT HAPPENS IF SRY PROTEIN IS INJECTED INTO A GENETIC FEMALE FETUS 6 WEEKS AFTER CONCEPTION
FEMALE WITH TESTES
WHAT HAPPENS IF DRUGS THAT BLOCK SRY PROTEIN ARE INJECTED INTO THE MALE FETUS
GENETIC MALE WITH OVARIES
WHEN DOES SEXUAL DEVELOPMENT OF INTERNAL REPRODUCTIVE DUCTS DEVELOP?
AT 6 WEEKS
WHAT DO BOTH SEXES BEGIN WITH
2 SETS OF REPRODUCTIVE DUCTS CALLED THE WOLFFIAN AND MULLIAN SYSTEM
WHAT IS THE WOLFFIAN SYSTEM
IT CONTAINS THE MALE INTERNAL ORGANS (SEMINAL VESICLES, VAS DEFERENS, (MALE REPRODUCTIVE DUCTS)
WHAT IS THE MULLIAN SYSTEM
IT CONTAINS THE INTERNAL FEMALE ORGANS ( UTERUS, VAGINA, FALLOPIAN TUBES
IS IT PHYSICALLY POSSIBLE TO HAVE 2 MALE AND 2 FEMALE ORGANS
NO BUT YOU CAN HAVE I TESTIS AND I OVAVRY
AT 6 WEEKS , ALL HUMAN FETUSES HAVE THE ANTECEDENTS OF
BOTH MALE (WOLFFIAN)
AND FEMALE (MILLERIAN) SYSTEMS
HOW IS THE WOLFFIAN SYSTEM DEVLELOPS AND (TIME)
UNDER THE INFLUENCE OF TESTICULAR TESTOSTERONE
AND MULLERIAN INHIBITING SUBSTANCE CAUSES THE MULLERIAN SYSTEM TO DEGENERATE
HOW DOES THE MULLERIAN SYSTEM DEVELOPS?
IN THE ABSENCE OF TESTOSTERONE, THE MULLERIAN SYSTEM DEVELOPS INTO FEMALE REPRODUCTIVE DUCTS AND THE WOLFFIAN SYSTEM FAILS TO DEVLOP
WHEN IS THERE A DIFFERENTIATION OF DUCTS
3RD PRENATAL MONTH OR 12 WEEKS
IN THE 3RD MONTH OF MALE FETAL DEVELOPMENT
THE TESTES SECRET TESTOSTERONS AND MULLERIAN INHIBITING SUBSTANCE AND THE TESTOSTERONE STIMULATES THE DEVELOPMENT OF THE WOLFFIAN SYSTEM AND THE MULLERIAN-INHIBITING SUBSTANCE CAUSES THE MULLERIAN SYSTEM TO DEGENERATE AND THE TESTES TO DESCENT INTO THE SCROTUM
WHAT IS SCROTUM
THE SAC THAT HOLDS THE TESTES OUTSIDE THE BODY CAVITY
WHAT HAPPENS IF GENETIC FEMALE IS INJECTED WITH TESTOSTERONE DURING THE APPROPRIATE FETAL PERIOD?
MALE REPRODUCTIVE DUCTS ARE DEVELOP ALONG WITH FEMALEONES
THE DIFFERENTIATION OF THE INTERNAL DUCTS OF THE FEMALE REPRODUCTIVE SYSTEM IS WHAT?
NOT UNDER THE CONTROL OF OVARIAN HORMONES, THE OVARIES ARE ALMOST COMPLETELY INACTIVE DURING FETAL DEVELOPMENT
THE DEVLOPEMENT OF THE MULLERIAN SYSTEM OCCURS IN ANY FETUS THAT IS NOT?
EXPOSED TO TESTICULAR HORMONES DURING THE CRITICAL FETAL PERIOD
WHEN DOES THE DIFFERENTIATION OF THE EXTERNAL GENITALIA OCCUR?
IN THE 3RD PRENATAL MONTH
THE DEVELOPMENT OF MALE AND FEMALE EXTERNAL REPRODUCTIVE ORGANS COME FROM WHAT?
THAT SAME BI-POTENTIAL PRECURSOR
WHAT IS THE DEVELOPMENT OF THE EXTERNAL GENITALS CONTROLLED BY
THE PRESENCE OR ABSENCE OF TESTOSTERONE

IF TESTOSTERONE PRESENT AT THE APPROPRIATE STAGE OF FETAL DEVELOPMENT YIELDS MALE EXTERNAL GENTILES FROM THE BI-POTENTIAL PRECURSOR
AND IF TESTOSTERONE NOT PRESENT
DEVELOPMENT OF THE EXTERNAL GENITALS PROCEEDS ALONG FEMALE LINES
WHAT HAPPENS IF THE TIMING IS OFF
for the possibility of mismatches between internal and external genitalia
WHAT IS Androgen Insensitivity Syndrome
produces XY individuals appear female because their bodies fail to listen to the androgen produces, these individuals appear to be female but have functioning testes
NORMALLY HOW DE WE JUDGE SEX
sex based on external genital
DURING CHILDHOOD WHAT IS LOW
LEVELS OF CIRCULATING GONADAL HORMONES ARE LOW

REPRODUCTIVE ORGANS ARE IMMATURE

AND MALES AND FEMALES ARE NOT DIFFERENT IN GENERAL APPEARANCE
WHAT IS PUBERTY
THE TRANSITIONAL PERIOD BETWEEN CHILDHOOD AND ADULTHOOD
WHAT HAPPENS IN PUBERTY
PRODUCTION OF HORMONES THAT
ESTABLISHES FERTILITY

GROWTH SPURT OCCURS

produces secondary sex characteristics
SECONDARY SEX CHARACTERISTICS
FEATURES OTHER THAN REPRODUCTIVE ORGANS THAT DISTINGUISH SEXUALLY MATURE MEN AND WOMEN
PUBERTY IS ASSOCIATED WITH AN INCREASE OF WHAT
GROWTH HORMONE

GONADOTROPIC HORMONE

ADRENOCORTICOTROPIC HORMONES
WHAT RELEASES THE HORMONES?
ANTERIOR PITUITARY
WHAT HORMONE IS THE ONLY ANTERIOR PITUITARY HORMONE THAT DOES NOT HAVE A GLAND AS IT PRIMARY TARGET
GROWTH HORMONE
GROWTH HORMONE
ACT DIRECTLY ON BONE AND MUSCLE TISSUE TO PRODUCE GROWTH
WHAT DOES THE GONADOTROPIC
AND ADRENOCORTICOTROPIC HORMONE DO
CAUSE THE GONADS AND THE ADRENAL HORMONES WHICH INITIATE THE MATURATION OF THE GENITALS AND THE DEVELOPMENT OF SECONDARY SEX CHARACTERISTICS
WHAT IS MASCULINIZATION
IN PUBERTAL MALES, ANDROGEN LEVELS ARE HIGHER THAN ESTROGEN LEVELS
WHAT CAUSES FEMINIZATION
IN PUBERTAL FEMALES, THE ESTROGEN ARE PREDOMINATE
WHAT HAPPENS IF INDIVIDUALS RE CASTRATED BEFORE PUBERTY?
DO NOT BECOME SEXUALLY MATURE AND NEED REPLACEMENT INJECTIONS OF ANDROGEN AND ESTROGEN
WHAT IS ANDROSTENEDIONE?
AN ANDROGEN THAT IS RELEASES BY THE ADRENAL CORTEX AND IS RESPONSIBLE FOR PUBIC HAIR AND AXILLARY HAIR IN FEMALES
WHY IS PUBERTY ACCELERATED
IMPROVEMENTS IN DIETARY, MEDICAL, AND SOCIOECONOMIC CONDITIONS
WHAT HAPPENS TO INDIVIDUALS WHO FAIL TO UNDERGO PUBERTY
DO NOT DEVELOP SEXUAL INTEREST
Hormones may affect continues sexual interest but are more important for
sexual performance especially men more than women
(hormones may be lost
to aging,
fertility decrease
in both men and women)
(fertility at
its height is between 20 and 30)
Hormone levels
are not related to sexual orientation
INTER SEX CONDITIONS
Having a mixture of male and female
Can’t have a full set of both
Congenital adrenal hyperplasia
leads to XX individuals having ambiguous external genitalia (but normal internal)
Androgen insensitivity syndrome leads
to development of testes but no other male internal or external reproductive organs
WHY ARE MEN BRAINS LARGER THAN WOMEN
DUE TO NUMEROUS ANATOMICAL DIFFERENCES

VOLUMES OF STRUCTURES

SIZES OF FIBER TRACTS

AND THE TYPE OF CELLS THAT MAKE UP STRUCTURES AND NUMBER AND TYPES OF SYNAPSES THAT CONNECT CELLS WITH STRUCTURES


BY 15 %
WHAT MASCULINIZES THE BRAIN
SEX STEROIDS ARE DERIVED FROM CHOLESTEROL AND CONVERTED FROM ONE TO THE OTHER AROMATIZED TESTOSTERONE BECOMES ESTRADIOL (FOUND IN RODENTS)
WHAT INTERFERES WITH MASCULINIZING EFFECTS OF TESTOSTERONE
BLOCKING AROMATIZATION OR ESTRADIOL RECEPTORS
WHY DOESN'T THE FEMALE BRAIN GET MASCULINIZE
IT WAS FOUND IN FEMALE RODENTS THAT

ALPHA FETOPROTEIN IN THE BLOOD DURING PREINATAL PERIOD PROTECTS THE FEMALE BRAIN FROM ESTRADIOL
WHY IS ESTRADIOL NOT BROKEN DOWN IN THE BRAIN
BECAUSE ALPHA FETO PROTEIN DOES NOT CROSS THE BLOOD BRAIN BARRIER
WHAT DO PERINATALHORMONES DO TO THE BEHAVIORAL DEVELOPMENT OF MALE AND FEMALE (RATS)
FOR MALES MASCULINIZE PROMOTES MALE BEHAVIOR AND DEFEMINIZE PREVENTING FEMALE BEHAVIOR
WHAT HORMONE MASCULINIZES AND DEFEMINIZES
PERI NATAL TESTOSTERONE
SIX WEEKS AFTER CONCEPTION, THE SYE GENE ON THE Y CHROMOSOME OF THE HUMAN MALE TRIGGERS THE PRODUCTION OF WHAT
SRY PROTEIN
IN THE ABSENCE OF SRY PROTEIN THE CORTICAL CELLS OF THE PRIMORDIAL GONADS DEVELOP INTO WHAT
OVARIES
IN THE 3RD MONTH OF MALE FETAL DEVELOPMENT, THE TESTES SECRETE ____________ AND __________________
TESTOSTERONE
AND INHIBITING MULLERIAN SUBSTANCE
THE HORMONAL FACTOR THAT TRIGGERS THE DEVELOPMENT OF HUMAN MULLERIAN SYSTEM IS ________
ANDROGEN OR TESTROGERN
_______ AND ____________ DEVELOP FROM THE SAME BIPOTENTIAL PRECURSOR
SCROTUM AND LABIA MAJORA
WHAT CAUSES THE BEHAVIORAL DIFFERENCES BETWEEN MEN AND WOMEN
PRENATAL ANDROGEN
HOW DOES ESTRADIOL GET INTO THE MALE BRAIN
TESTOTERON ENTERS THE BRAIN AND IS CONVERTED TO ESTRADIOL
WHAT DETERMINES IF MALE OR FEMALE DEVELOP
LEVELS OF ANDROGEN AND ESTROGEN
WHAT DISEASE IS WHERE THE LITTLE GIRL GREW INTO A BOY
CONGENITAL ADRENAL HYPERPLASIA
THE WOMEN WHO WASN'T
ANDROGEN INSENSITIVITY SYNDROME
WHY WAS ANNE A MAN
XY CHROMOSOME

INTERNALIZED TESTES BUT NO OVARIES
hORMONE TEST WERE LEVELS OF A MAN
WHAT CAUSES ANDROGENIC INSESITIVITY SYNDROME
MUTATION IN THE ANDROGEN RECEPTOR GENE THE ANDROGEN RECEPTORS ARE UNRESPONSIVE
DURING DEVELOPMENT ANNE'S TESTES RELEASED
NORMAL ANDROGENS FOR A MALE BUT HER BODY COULD NOT RESPOND TO THEM BECAUSE OF THE MUTATION
WHAT HAPPENS WHEN ANN GETS TO PUBERTY
AT PUBERTY

ANNES TESTES RELEASED ENOUGH ESTROGEN TO FEMINIZE HER BODY IN THE ABSENCE OF THE COUNTERACTING EFFECTS OF ANDROGEN

HER ADRENAL ANDROSTENEDIONE WAS NOT STIMULATED THE GROWTH OF PUBIC AND AXILLARY HAIR
WHAT HAPPEN DURING FETAL DEVELOPMENT TO ANN BODY
NORMAL INTERNAL FEMALE REPRODUCTIVE DUCTS DURING FETAL DEVELOPMENT

SHE HAD RELEASED NORMAL AMOUNTS OF ANDROGENS FOR A MALE BUT HER BODY DID NOT RESPOND TO THE ANDROGEN AND SO HER BODY RESPONDED LIKE A FEMALE

SHE HAD FEMALE EXTERNAL GENITAL AND HER BRAIN AND BEHAVIOR DEVELOP LIKE A FEMALE
WHO DOES ONE DEVELOP ADRENOGENITAL SYNDROME
CAUSED BY CONGENITAL ADRENAL HYPERPLASIA
ADRENOENITAL SYNDROME AFFECT BOYS
LITTLE EFFECT ON THE DEVELOPMENT OF MALES BUT
ACCELERATES PUBERTY
ADRENONITAL SYNDROME FOR GIRLS
DX AT BIRTH
ABNORMALITIES OF THE EXTERNIAL GENITALS ARE CORRECTED AT BIRTH


ENLARGE CLITORIS AND PARTIALLY FUSES LABIA