Bccc Ormc Renal

Front 1

The filtering point for fluid, nutrients and waste products contained in blood entering the kidney is called the

Back 1

Glomerulus

Front 2

Dilution or concentration of filtrate occurs in this section of the nephron

Back 2

Loop of Henle

Front 3

The functional units of the kidney is the

Back 3

Nephron

Front 4

This area of the nephron serves as a holding area for the filtrate before it moves through the proximal tubule

Back 4

Bowman's capsule

Front 5

The Juxtamedullary nephrons

Back 5

Are primarily responsible for concentration and dilution of urine

Front 6

Osmosis is the movement of water from an area of high solute concentration to an area of lower solute concentration

Back 6

False

Front 7

The movement of potassium from the blood into the Renal tubules for excretion usually involves active transport

Back 7

True

Front 8

What factor does not affect the level of urea production. Protein intake, volume status, blood pressure, infection

Back 8

Infection

Front 9

Which hormones play a role in maintaining fluid balance in the body

Back 9

Antidiuretic hormone, aldosterone, atrial natriuretic peptide

Front 10

Of the 180 L of infiltrate that are formed daily, what percent is excreted as urine

Back 10

1

Front 11

Tubular secretion of excess substances from the blood into the tubules is

Back 11

The last step in the formation of urine

Front 12

Tubular secretion plays a lesser roll in tubular reabsorption in changing filtrate into urine

Back 12

True

Front 13

Tubular reabsorption in the kidney occurs only by active transport

Back 13

False

Front 14

When pressure in the afferent arteriole decreases the -- is activated resulting in constriction of the Efferent arteriolar thereby increasing the GFR

Back 14

Activation of the RAAS

Front 15

– Is the mechanism by which Renal bloodflow and GFR remains constant despite changes in MAP

Back 15

Intrarenal autoregulation

Front 16

Renal bloodflow is regulated through three mechanisms. These are

Back 16

Intrarenal autoregulation, the sympathetic nervous system and the RAAS

Front 17

Which EKG change may be a result of hypocalcemia

Back 17

Prolongation of QTinterval

Front 18

Calcium and phosphorus have an inverse relationship

Back 18

True

Front 19

Neuromuscular signs of hyper Kaylee me up include

Back 19

Numbness and tingling of the face, hands and tongue

Front 20

Management of normal potassium values are significant in preventing organ system dysfunction such as

Back 20

Cardiovascular abnormalities

Front 21

Patients with water intoxication will exhibit

Back 21

Confusion, anorexia, decreased serum osmolality

Front 22

Patients with saline excess will present with

Back 22

Dyspnea, bounding pulses, diaphoresis

Front 23

Patients with saline deficit will present with

Back 23

Tachycardia, decreased urine output, renal failure

Front 24

What are three signs and symptoms of hypomagnesemia

Back 24

Respiratory depression, dysrhythmias, and confusion

Front 25

Which of the following EKG changes may be a result of hypokalemia

Back 25

Ventricular ectopy

Front 26

The etiology or clinical presentation of hyperphosphatemia include

Back 26

Chronic kidney failure, muscle tetany, soft tissue calcification

Front 27

Chvostecks sign and Troussous sign indicate

Back 27

Hypocalcemia

Front 28

Alkalosis, hypoparathyroidism, pancreatitis, and hypomagnesium may all be associated with

Back 28

Hypocalcemia

Front 29

The patient with chronic kidney failure is at risk to develop osteoporosis due to which electrolyte imbalance

Back 29

Hyperphosphatemia

Front 30

When teaching the patient about the consequences of chronic kidney failure, the nurse explains that the altered bone metabolism occurs as a result of

Back 30

Elevated serum phosphorus level

Front 31

The nurse instructs the patient with chronic kidney failure to eat

Back 31

Low protein, low potassium, low-sodium, high calcium

Front 32

When assessing a patient's risk factors for chronic kidney failure, the nurse would be most concerned when the patient reports

Back 32

Poorly controlled diabetes