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74 Cards in this Set
- Front
- Back
Amyl nitrate
use |
relief of acute anginalpain in adults
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Isosorbidemononitrate(ISMO, Imdur)
uses |
prevention and treatment of angina in adults
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Nitroglycerin (Nitro-Bid, Nitrostate)
uses |
treatment of acute angina attack; prevention of anginalattacks
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Beta Blockers
•Action |
–Block beta-adrenergic receptors in the heart and kidneys, decreasing the influence of the SNS on these tissues; decrease cardiac output and the release of renin
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Beta Blockers
•Indications |
–Treats stable angina pectoris and hypertension; prevents reinfarctionin MI patients; treats stable CHF
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Beta Blockers
Pharmacokinetics |
–Absorbed in the GI tract, undergoes hepatic metabolism, and is excreted in the urine
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Beat Blockers
•Contraindications |
–Bradycardia
–Heart block –Cardiogenicshock –Asthma and COPD –Pregnancy and lactation |
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Beta Blockers
•Cautions |
–DM
–PVD –Thyrotoxicosis |
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Beta Blockers
Adverse Effects |
–Related to their blockage of sympathetic nervoucsystem
–CNS: dizziness, fatigue, and emotional depression –GI: N/V, colitis –CV: CHF, decreased cardiac output, arrhythmias –Bronchospasm, dyspnea, cough |
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Beta Blockers
•Drug-to-drug interaction |
–Clonidine
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•Metoprolol(Toprol, ToprolXL)
Uses |
treatment of angina in adults, prevention of reinfarctionwithin 3-10 days after MI
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Nadolol(Corgard)
uses |
long term management of angina in adults
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Propranolol(Inderal)
uses |
long term management of angina and prevention of reinfarctionin patients 1 -4 weeks after MI in adult
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Calcium Channel Blockers
•Actions |
Inhibit the movement of calcium ions across the membranes of myocardial and arterial muscle cells, altering the action potential and blocking muscle cell contraction
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Calcium Channl Blockers
•Indication – |
Prinzmetal’sangina
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CCB
Pharmocokenetics |
–Well absorbed
–Metabolized in the liver and excreted in the urine |
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CCB
Contraindications |
–Allergy
–Heart block and sick sinus syndrome –Renal or hepatic dysfunction –Pregnancy and lactation |
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Calcium Channel Blockers
•Adverse Effects |
–Hypotension
–Cardiac arrhythmias –GI upset –Skin reaction –Headache •Drug-to-drug interactions –Vary with each drug |
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Amlodipine(Norvasc),
uses |
treatment of chronic, stable angina and of Prinzmetal’sangina in adults
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Diltiazem(Cardizem, CardizemSR)
uses |
treatment of angina in adults
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Nicardipine(Cardene)
uses |
treatment of angina in adults
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Nifedipine(Adalat, Procardia)
uses |
treatment of angina in adults
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Verapamil(Calan, Isoptin)
|
treatment of angina in adults.
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Risk Factors
CAD |
elevated blood lipid levels smoking
hypertension diabetes mellitus obesity family history of premature cardiovascular disease (first-degree relative with cardiovascular disease at 55 years of age or younger for men and at 65 years of age or younger for women) age> 45 years for men; >55 years for women. |
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CAD
Prevention (modifiable) |
cholesterol abnormalities tobacco use
hypertension diabetes mellitus Lack of estrogen in women Obesity Physical inactivity |
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CAD
non mosdifiable risk factors |
Family history of coronary heart disease
Increasing age Gender (men develop CAD at an earlier age than women) Race (higher incidence of heart disease in African Americans than in Caucasians) |
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LDL Goal
for patients with one or no risk factors |
Less than 160 mg/dL
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LDL Goal
for patients with two or more risk factors |
Less than 130 mg/dL
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LDL goal
patients with CAD or at high risk for CAD. |
Less than 100 mg/dL
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LDL Goal
patients at very high risk for an acute coronary event |
Less than 70 mg/dL is
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HDL Goal
|
> 40 mg/dL and should ideally be > 60 mg/dL.
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Triglycerides
|
< 200
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Stable angina:
|
predictable and consistent pain that occurs on exertion and is relieved by rest
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Unstable angina (also called preinfarction angina or crescendo angina):
|
symptoms occur more frequently and last longer than stable angina. The threshold for pain is lower, and pain may occur at rest
Unstable angina is characterized by attacks that increase in frequency and severity and are not relieved by rest and nitroglycerin. Patients with unstable angina require medical intervention. |
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Intractable or refractory angina:
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severe incapacitating chest pain
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Variant angina (also called Prinzmetal's angina):
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pain at rest with reversible ST-segment elevation; thought to be caused by coronary artery vasospasm
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Silent ischemia:
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objective evidence of ischemia (such as ECG changes with a stress test), but patient reports no symptoms
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CC Blockers
ex |
(Norvasc) and diltiazem
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MI
EKG changes |
The classic ECG changes are T-wave inversion, ST-segment elevation, and development of an abnormal Q wave
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PTCA
|
invasive interventional procedure, a balloon-tipped catheter is used to open blocked coronary vessels and resolve ischemia
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The major indications for CABG
|
Alleviation of angina that cannot be controlled with medication or PCI
Treatment of left main coronary artery stenosis or multivessel CAD Prevention and treatment of MI, dysrhythmias, or heart failure Treatment for complications from an unsuccessful PCI |
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Post op chest tube amoutns
|
. Bloody drainage should not exceed 200 mL/h for the first 4 to 6 hours post op
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postpericardiotomy syndrome:
s/s |
fever, malaise, pericardial effusion, pericardial friction rub, arthralgia
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Right-sided failure
def |
–RV cannot eject sufficient amounts of blood, and blood backs up in the venous system.
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Left-sided failure
def |
LV cannot pump blood effectively to the systemic circulation. Pulmonary venous pressures increase,
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Right-sided failure
s/s |
edema, hepatomegaly, ascites, anorexia, nausea, weakness, and weight gain.
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Left-sided failure
s/s |
pulmonary congestion with dyspnea, cough, crackles, and impaired oxygen exchange.
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Classification of Heart Failure
|
NYHA classification of HF
–Classification I, II, III, IV •ACC/AHA classification of HF –Stages A, B, C, D •Treatment guidelines are in place for each stage. |
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Nursing Process:
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The Care of the Patient with HF: Planning
•Goals may include promoting activity and reducing fatigue, relieving fluid overload symptoms, decreasing anxiety or increasing the patient’s ability to manage anxiety, encouraging the patient to make decisions and influence outcomes, teaching the patient about the self-care program. |
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Orthopnea
|
difficulty breathing when lying flat, is a clinical manifestation of left-sided heart failure.
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Pulmonary Edema
def |
•Acute event in which the LV cannot handle an overload of blood volume. Pressure increases in the pulmonary vasculature, causing fluid to move out of the pulmonary capillaries and into the interstitial space of the lungs and alveoli.
•Results in hypoxemia |
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Pulmonary Edema
s/s |
Clinical manifestations: restlessness, anxiety, dyspnea, cool and clammy skin, cyanosis, weak and rapid pulse, cough, lung congestion (moist, noisy respirations), increased sputum production (sputum may be frothy and blood-tinged), decreased level of consciousness
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HF
Class1 |
No limitation of physical activity
Ordinary physical activity does not cause undue fatigue,palpitations or dyspnea |
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HF
Clss2 |
slight limitation of physical actvitiy. Comfortable at rest, but ordinary physical activity results in fatigue, palpiation or dyspnea.
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HF
Class 3 |
Marked limitation of physical activity. Comfortable at rest., but less than ordinary activity causes fatigue, palpiation or dyspnea.
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HF
Class 4 |
Unable to carry out any physical activity without discomfort. Symptoms of cardia insufficiency at rest. If any ohysical activity, discomfort is increased.
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afterload
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the amount of resistance to ejection of blood from a ventricle
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urine output of less than 50 mL per 24 hours
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anuria
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the amount of blood pumped out of the heart in 1 minute
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cardiac output (CO)
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a treatment for heart failure in which a device paces both ventricles to synchronize contractions
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cardiac resynchronization therapy (CRT)
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contractility
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the force of ventricular contraction; related to the number and state of myocardial cells
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ejection fraction (EF)
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percentage of blood volume in the ventricles at the end of diastole that is ejected during systole; a measurement of contractility
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the inability of the heart to pump sufficient blood to meet the needs of the tissues for oxygen and nutrients; signs and symptoms of pulmonary and systemic congestion may or may not be present
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heart failure (HF)
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implantable cardioverter defibrillator (ICD)
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a device implanted in patients with ventricular dysrhythmias that detects and treats dysrhythmias
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left-sided heart failure (left ventricular failure)
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inability of the left ventricle to fill or pump (empty) sufficient blood to meet the needs of the tissues for oxygen and nutrients; traditional term used to describe patient's symptoms of heart failure
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oliguria
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diminished urine output; less than 400 mL per 24 hours
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orthopnea
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shortness of breath when lying flat
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preload
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the amount of myocardial stretch just before systole caused by the volume of blood presented to the ventricle
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pulseless electrical activity (PEA)
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condition in which electrical activity is present but there is not an adequate pulse or blood pressure because of ineffective cardiac contraction or circulating blood volume
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pulsus paradoxus
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systolic blood pressure of more than 10 mm Hg higher during exhalation than during inspiration; difference is normally less than 10 mm Hg
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right-sided heart failure (right ventricular failure)
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inability of the right ventricle to fill or pump (empty) sufficient blood to the pulmonary circulation
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stroke volume (SV)
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amount of blood pumped out of the ventricle with each contraction
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systolic heart failure
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inability of the heart to pump sufficiently because of an alteration in the ability of the heart to contract; current term used to describe a type of heart failure
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Cardiac tamponade
s/s |
feeling of fullness within the chest
substantial or ill-defined pain pressure in the chest may result from stretching of the pericardial sac. venous pressure increased engorged neck veins shortness of breath labile or low blood pressure. pulsus paradoxus falling systolic blood pressure narrowing pulse pressure rising venous pressure (increased JVD distant (muffled) heart sounds |