Acute And Chronic Reanl Failure

Front 1

characteristics of acute RF

Back 1

sudden onset
approx 50% of nephrons involved
2-3 week duration
good prognosis for return to norm. high mortality in some situations

Front 2

characteristics of Chronic RF

Back 2

gradual onset
90-5% involvement
permanenet
fatal wo dialysis, transplant

Front 3

decreased urine specific gravity

Back 3

indicates loss of urine concentrating ability. earliest sign of renal tubule damage

Front 4

kidney response to hypovoemic shock

Back 4

RAAS activation, constriction of renal blood vessel constriction, rlease of ADH

Front 5

salt substitutes and renal failure

Back 5

pt. should aovif as many contain potassium

Front 6

ARF and anemia

Back 6

not a problem like in CRF

Front 7

Assessing dialysis effctiveness

Back 7

Serum Cr is used

Front 8

Urine spcific gravity, pH norms

Back 8

specific gravity=1.010-1.025
pH=5.5-6

Front 9

IV fat emulsion in RF therapy

Back 9

good non-protein source of claories

Front 10

renagel~ can I chew it?

Back 10

must take whole

Front 11

calcium carbonate in RF therapy

Back 11

give after meals as phosphate binder

Front 12

uremic syndrome

Back 12

systemic and clinical manifestations of ESRD

Front 13

renal dose dopamine

Back 13

weigh accurately to determine dosage, if infiltration, stop infusion but do not d/c iv, use to infuse antidote, phentolamine

Front 14

hyposhtenuria

Back 14

inability to concentrate urine

Front 15

PTH

Back 15

controls amount of phosphate in blood. causes excretion if excess. chronic hypocalcemia cuses constant stimulation of Parathyroid, leading to release of calcium from bones.

Front 16

BUN pedi

Back 16

newborn 4-18
infant/child= 5-18

Front 17

Creatinine pedi

Back 17

newborn= 0.2-0.4
infant/child= 0.3-0.7
adolescent= 0.5-1.0

Front 18

minimal change nephrotic syndrome

Back 18

same as idiopathic nephrosis