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63 Cards in this Set

  • Front
  • Back
PQRST model of symptom analysis
P-what was the PRVOKING INCIDENT the caused the pain, if any?

Q-What is the QUALITY of the pain? Is it burning,throbbing, stabbing?

R-Where is the REGION of the pain? Does it RADIATE? Does anything RELIEVE the pain?
S-How SEVERE is the pain?

T-What is the TIMING of the pain? When does it occur and how long does it last?
CMS neurovascular assessment
Circulation
Movement
Sensation
(skin color, temperature, mvmt, sensation, pulses, capillary refill, pain)

especially important when external devices such as casts and bulky dressings can compress compartments created by sheaths if inelastic fascia, causing extensive tissue damage
Body alignment assessment
observe posture/positioning while sitting, lying,standing

assess for scoliosis and kyphosis
gait assessment
observe while client ambulates if client can walk

notice stance and swing phases
joint assessment
head to toe

proper alignment
symmetry
redness
swelling
ROM for function and crepitus
skeletal muscle assessment
examine at same time as joints
observe each major muscle group for symmetry in:
size
shape
tone
strength
assess neurovascular status:
skin color
inspect area distal to injury

normal is no change compared with other parts of body
assess neurovascular status:
skin temperature
palpate area distal to injury
skin should be warm
assess neurovascular status:
movement
ask client to move affected area or area distal to injury

normal-client can move w/o discomfort
assess neurovascular status:
sensation
ask if numbness/tingling present
palpate w/safety pin or paper clip, esp. the web space between first and second toes or between thumb and forefinger

normal is no numbness or tingling, no difference in sensation in affected& unaffected extremities

loss of sensation in these areas idicates peroneal nerve or median nerve damage
assess neurovascular status:
pulses
palpate distal to injury
normal is pulse that is strong and easily palpated; no difference in affected/unaffected extremities
assess neurovascular status:

capillary refill
press nail bed distal to injury until blanching

normal is blood return within 3 seconds, 5 seconds for older adult
assess neurovascular status:
pain
ask about location, nature, frequency of pain
CT computed tomography
provides better picture of soft tissues and less-dense bone than x-rays, esp. in vertebral column

can identify CNS probs., strokes/tumors

may be done with or w/o contrast

If contrast used, make sure NPO for 4 hours, no allergy to iodine or seafood, signed informed consent form
x-ray
detect bone density,swelling, alignment, continuity
MRI
often more accurate than x-ray or CT for detecting soft tissue damage

contrast may be used
ask pt. to remove any metal objects

joint implant safe

pacemakers not safe
arthrogram
enhanced radiogram of joint obtained after contrast injected

most common for knees and shoulders

allergies to iodine/seafood?

joint swelling caused by injection fluid will diminsh w/in 1-2 days
usual activities resume w/in 24 h.
myelogram
radiograph of vertebral spine obtained after injecting contrast into lumbar subarachnoid space

can visualize spinal cord, vertebral bones, intervertebral disks, surrounding soft tissue

after test, client must be properly positioned to prevent CSF leakage and headaches
arthrocentesis
diagnostic or treatment

sample of synovial fluid withdrawn and sent to lab for analysis

excess fluid can be removed
alkaline phosphatase
enzyme that tends to increase when bone or liver damaged

increase in serum levels reflect increase in osteoblastic activity
quadriplegia
paralysis of arms,legs, trunk below level of injury
quadriparesis
numbness or other abnormal or impaired sensation in all four limbs and trunk
paraplegia
paralysis characterized by motor or sensory loss in legs and trunk
paraparesis
numbness or other abnormal or impaired sensation in legs and trunks
hemiplegia
paralysis of one side of body
hemiparesis
numbness or other abnormal or impaired sensation on one side of body
kyphosis
increased convexity in the throacic spine when viewed from side

shoulder slouched

vertebral bones prominent

make sure not interfering w/ breathing
crepitus
grating sound caused by joint deterioration
spastic
muscle contraction caused by reflex rather than by CNS control
flaccid
state of being weak, soft, flabby, lacking normal muscle tone, having no ability to contract
proprioception
sensation pertaining to stimuli originating from within the body regarding spatial position and muscular activity or to the sensory receptors that they activiate
isotonic
form of active exercise in which muscle contracts and move with little change in resistance
isometric
increase muscle tension by applying pressure against stable resistance where there is no joint mvmt and the length of the muscle remains unchanged, but the tone and strength are maintained or increased
Diagnosis:
Impaired physical mobility
goal/expected outcome
overall goal is to improve mobility and prevent complications of immobility

move independently in wheelchair

transfer independently from bed to chair using sliding board

performs ADLs independently using assitive or adaptive devices

maintains intact skin
Diagnosis:
activity intolerance
goal/expected outcome
overall goal is to improve endurance and tolerance to activities

client tolerated activity w/o evidence of dizziness, fatigue, weakness, abnormal VS, ECG changes, exertional discomfort, or dyspnea
disuse
decrease or cessation of use of an organ or body part, restriciton of activity or immobility
immobility
inablility to move whole body or body part
bed rest
prescribed or self-imposed restriction to bed for therapeutic reasons
atrophy
decrease in size or physiological activity of normally developed tissue or organ as result of inactivity or diminished function

after 24-36 hours of inactivity, muscle begins to lose contractile strength
contracture
abnormal condition of joint flexion resulting from shortening of muscle fiber and associated connective tissue, with resistance to stretching and eventually flexion and finally to permanent fixation

can result from loss of normal skin elasticity as in scar formation
footdrop
contracture deformity in which muscles on anterior lengthen. At same time, muscles of plantar flexion and achilles tendon shorten, resulting in plantar flexion of foot
osteoporosis
condition in which there is a decreaed mass per unit volume of normally mineralized bone, primarily from a loss of calcium, that makes bones brittle and porous
shearing force
mechanical force that acts on an area of skin in a direction parallel to the body's surface.

shear injuries are serious form of pressure injury because they result in necrosis and ulceration
excoriation
injury to epidermis caused by abrasion, scratching, burn, chemicals such as sweat, wound drainage, feces or urine coming in contact w/ skin
maceration
softening of epidermis caused by prolonged contact w/ moisture, such as from a wet sheet or diaper

increases risk for damage by decreasing skin's ability to resist trauma
orthostatic hypotension
drop in systolic BP of 20mmHg or more and drop in diastolic BP of 10mmHg or more for 1 or 2 minutes after moving to standing position
deep vein thrombosis
blood clot develops in lumen of deep leg vein, such as tibial, popliteal, femoral or iliac vein
hypostatic pneumonia
inflammation of lungs caused by stasis of secretions, which become medium for bacterial growth

signs are thickened yellow sputum, ronchi, crackles, wheezes, fever, rapid shallow breathing
pulmonary emboli
piece of thrombus that breaks free, carried by veins to right heart and pulmonary circulation, lodges in pulmonary blood vessel

blood flow and o2 can't reach area of lung tissue served by the blocked vessel

S &S: sudden onset of dyspnea, cough, sudden chest pain, hemoptysis, tachycardia, tachypnea

emergency
renal calculi
stones formed in kidney when excretion rate of calcium or other minerals is high, as when osteoclastic activity releases calcium from bones during immobility

exacerbated because end products of metabolism in inactive client are generally alkaline because of decreased muscle activity

occur after only 1-2 weeks of inactivity
muskuloskeletal effects of immobility
deconditioning

atrophy

body breaks down muscle mass to obtain energy, can result in negative nitrogen balance

more susceptible to ambulation and falls

after 5 days, calcium and fibrotic cells deposit joints causing them to become stiff and lose range of motion

contractures

fibrosis

footdrop

osteoporosis
integumentary effects of immobility
pressure ulcer
shear
friction injury
excoriation
maceration
cardiovascular effects of immobility
oxygen demand on cells decrease, cardiovascular workload may increase

deconditioning

workload on heart increases when pt changes position or performs ADLs

valsalva maneuver

orthostatic intolerance

edema



DVT

pulmonary embolus
respiratory effects of immobility
prolonged bed rest has negative effects on resp function

recumbent position compromises respiratory function and predisposes pt to resp. complications, hypoventilation, atelectasis, stasis of secretions, altered gas exchange

resp. muscles weaken, decrease bellows effect

compromises ability to cough

hypostatic pneumonia
GI effects of immobility
slows BMR and GI motility, manifestd as anorexia, constipation, increased storage of fat and carbs, negative nitrogen balance

may become malnourished: anemia, albuminemia, decreased immunity, dry cracked skin

blood cells can't be synthesized w/o sufficient protein

not enough WBC= lowered immunity

not enough RBC's=fatigue, anemia, poor wound healing

skin, nails, hair become dry and brittle

loss of sub q fat affects ability to conserve heat and protect bony prominences

decreased fluid intake:
dehydration->
constipation
decreaed blood volume
decreased tissue perfusion
decreaed urine output
stasis of respiratory secretions
GU effects of immobility
diminishes kidney and bladder function

increased urinary stasis, retention, renal calculi, UTI's

UTI most prevalent hospital acquired infections

inadequate personal hygiene predisposes client to to UTI - reflux of urine can spread microorganisms to kidneys
factors affecting a client's ability or desire for mobility
pain

therapeutic or prescribed inactivity

change in LOC
change musculoskeletal function

emotional or psychological disturbances

chronic illnesses
assess pt at risk for disuse syndrome
primary risk factor: immobility


level of inactivity and duration of inactivity

other related factors
A-age
B-body weight
C-chronic illness
D-discomfort
E-environment
intervention to prevent skin breakdown
monitor client
cleanse skin
increase circulation
decrease microorganisms
remove excess moisture
decrease friction/excoriation
Provide nutrition
Position client properly
prevent shear
decrease pressure
Use support surfaces when indicated
intervention to maintain circulatory function
decrease edema formation
elevate
decrease blood stasis
TEDs
leg exercises
SCDs

maintain venous return
don't cross legs at knee
avoid tight socks

encourage correct breathing
prevent orthostatic intolerance
intervention to maintian respiratory function
encourage lung expansion
mobilize secretions
compartment syndrome
increased pressure w/in muscle compartment
symptoms of compartment syndrome
Acute Compartment Syndrome
The classic sign of acute compartment syndrome is pain, especially when the muscle within the compartment is stretched.

•The pain is more intense than what would be expected from the injury itself. Using or stretching the involved muscles increases the pain.
•There may also be tingling or burning sensations (paresthesias) in the skin.
•The muscle may feel tight or full.
•Numbness or paralysis are late signs of compartment syndrome. They usually indicate permanent tissue injury.

Chronic (Exertional) Compartment Syndrome
Chronic compartment syndrome causes pain or cramping during exercise. This pain subsides when activity stops. It most often occurs in the leg.

Symptoms may also include:

•Numbness
•Difficulty moving the foot
•Visible muscle bulging