Acp Essenttials Diabetes

Front 1

when to screen for DM

Back 1

when BP iss consisstently greater than 135/80

Front 2

random BG of what is predictive of DM

Back 2

200

Front 3

Fasting BG highly specific for DM

Back 3

126 or higher

Front 4

when to initiate ACE in DM

Back 4

the presscence of microalbuminuria

Front 5

target HbAIc

Back 5

less than 7

Front 6

what qualifies as imparied glucose tolerance after OGTT

Back 6

140 - 199 after OGTT (2hour)

fasting 100 - 125

Front 7

baseline creatinine formetformin

Back 7

lesss than 1.6mg/dl in men and 1.5/mg/dl in women

Front 8

used only in combo with sssulfonyl urea or ssulfonyl exenatide is what

Back 8

twice daily injectablemed that reduces HbAIc by .5 - 1 percent up to 40% of patients experience nausea

exenatide is an incretin mimetic that increases inssulin secretion, slows gastric emptying and decreases food intake

Front 9

what iss exenatide

Back 9

an incretin mimetic that slows gastric emptying, increasses insulin secretion and decreases food intake

Front 10

what iss exenatide

Back 10

an incretin mimetic that slows gastric emptying, increasses insulin secretion and decreases food intake

Front 11

2nd gen sulfonylureas

Back 11

gyburide glipizide

Front 12

2nd gen sulfonylureas

Back 12

gyburide glipizide

Front 13

1st gen. sulfonylureas

Back 13

tolbutamide, tolazimide, chlorpropramide

Front 14

1st gen. sulfonylureas

Back 14

tolbutamide, tolazimide, chlorpropramide

Front 15

how do ssulfonylureas work

Back 15

block ATP dependent K chanells causing depoarization of beta cellss which leads to inssulin releasse

Front 16

how do ssulfonylureas work

Back 16

block ATP dependent K chanells causing depoarization of beta cellss which leads to inssulin releasse

Front 17

how do ssulfonylureas work

Back 17

block ATP dependent K chanells causing depoarization of beta cellss which leads to inssulin releasse

Front 18

name the thiazilidinedioness and how they work

Back 18

Rosiglitazone and and pioglitazone and they work by binding to peroxissome proliferator activating receptor leading to increased insulin ssenssitization

Front 19

name the thiazilidinedioness and how they work

Back 19

Rosiglitazone and and pioglitazone and they work by binding to peroxissome proliferator activating receptor leading to increased insulin ssenssitization

Front 20

name the thiazilidinedioness and how they work

Back 20

Rosiglitazone and and pioglitazone and they work by binding to peroxissome proliferator activating receptor leading to increased insulin ssenssitization

Front 21

what ssecond gen ssulfonylurea should be decreased in kidney dysfxn

Back 21

glyburide

glypizide for hepaic

Front 22

ssside effects of metformin (biguanide)

Back 22

lactic acidosis, decreased B12, abnormaltase

Front 23

what diabetic drug decreases B12

Back 23

metformin

Front 24

dawn phenomenon

Back 24

rise in BG between 4-11am due to release of growht hormone, cortissol, glucagons and epinephrine

Front 25

SSomogyi effect

Back 25

reboubnd rise in morning glucose secondary to low overnight BG

Front 26

diabetic medss associated with increased Cardiovascular rissk

Back 26

the TZDs which are contraindicated in hear failure (pioglitazone, rossiglitazone)

Front 27

diabetic medss associated with increased Cardiovascular rissk

Back 27

the TZDs which are contraindicated in hear failure (pioglitazone, rossiglitazone)

Front 28

diabetic medss associated with increased Cardiovascular rissk

Back 28

the TZDs which are contraindicated in hear failure (pioglitazone, rossiglitazone)

Front 29

target preprandial glucose

Back 29

90 - 130

Front 30

target postprandial

Back 30

lesss than 180

Front 31

DKA dx triad

Back 31

pH less than 7.3, BG greater than 250, and ketoacidossis (bicarb less than 15 and possitive ketones)

Front 32

DKA dx triad

Back 32

pH less than 7.3, BG greater than 250, and ketoacidossis (bicarb less than 15 and possitive ketones)

Front 33

DKA dx triad

Back 33

pH less than 7.3, BG greater than 250, and ketoacidossis (bicarb less than 15 and possitive ketones)

Front 34

DKA therapy

Back 34

first infuse 0.9% NaCls along with IV insulin drip, once CBG is less than 250 start D5 or D10s and Q1hour K levels.

the intital inssulin boluss should be 0.15U/Kg then 0.1/Kg infusion

Front 35

DKA therapy

Back 35

first infuse 0.9% NaCls along with IV insulin drip, once CBG is less than 250 start D5 or D10s and Q1hour K levels.

the intital inssulin boluss should be 0.15U/Kg then 0.1/Kg infusio

Front 36

DKA therapy

Back 36

first infuse 0.9% NaCls along with IV insulin drip, once CBG is less than 250 start D5 or D10s and Q1hour K levels.

the intital inssulin boluss should be 0.15U/Kg then 0.1/Kg infusio

Front 37

DKA therapy

Back 37

first infuse 0.9% NaCls along with IV insulin drip, once CBG is less than 250 start D5 or D10s and Q1hour K levels.

the intital inssulin boluss should be 0.15U/Kg then 0.1/Kg infusio