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12 Cards in this Set

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PVT
>180bpm, very wide QRS complex
originates in VENTRICLE +PULSELESS
Ventricular Fibrillation
uncoordinated ventricular contractions
HYPOXIA primary cause, can lead to ischemia & asystole
Pulseless Electrical Activity PEA
ALWAYS CHECK FOR PULSE 1ST!
HYPOVOLEMIA primary cause, treat like asystole,
ASYSTOLE
true state of NO Electrical Activity
1st ° AV Block
PR interval > 0.20 sec
of no consequence UNLESS MI or electrolyte imbalance
2nd ° Heart Block type 1
Also called Mobitz 1, PR interval prolonged till atrial impulse blocked, thus, no QRS impulse
Atrial rhythm will be REGULAR
2nd ° Heart Block type 2
Mobitz II, almost always disease of distal/ventricular conduction
non conducted P waves, thus no QRS; no PR prolongation, wide QRS complex CAN LEAD TO COMPLETE BLOCK, txt= transcutaneous/transvenous pacing, or *Atropine
Complete Heart Block
3rd °, impulse generates in SA node, not conducted to ventricles
most common causes infarction & ischemia; txt= transcutaneous pacing; s&s= bradycardia, hypotension, hemodynamic instability
Supraventricular Tachycardia SVT
drop in cardiac output, HR >150 bpm
unstable, txt= cardioversion S&S= SOB, Palpitations, angina, dizziness, LOS, parasthesia, hyperventilation
Atrial Flutter
atrial contractions 240-350 bpm due to electrical activity in SA/AV node loop
S&S= palpitations, SOB,dizziness, nausea, impending doom, Peripheral edema, activity intolerance EARLY CARDIOVERSION
A FIB
SA node overwhelmed by atrial impulses, leads to irregular impulses to ventricles
no P -waves before QRS, HR is IRREGULAR, same S&S as A flutter
Other Tachycardias
monomorphic will deteriorate to V-Fib; polymorphic originates in ventricles rather than normal rhythm (from atria)