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56 Cards in this Set

  • Front
  • Back
Abdominal vasculature exams
renal artery-vein/ mesenteric arteries/ portal venous system/ hepatic vasculature/ abdominal aorta for aneurysms-stenosis
What can fixed hypertension be caused by?
chronic renal disease/ renal artery disease/ true renovascular hypertension
Renovascular hypertension etiology
atherosclerosis (most common)/ fibromuscular dysplasia (FMD)/ dissection and/or extension of aortic dissection
Indications for renal doppler exam
uncontrolled hypertension, especially in younger pts/ decreasing renal function/ abdominal bruit
Main renal arteries arise from ____ just distal to the ____.
aorta/ sma
Right renal artery courses ____ to the IVC.
posterior
Right renal artery is ____ than the left renal artery.
longer
Left renal vein lies ____ to the SMA and ____ to the aorta.
posterior/ anterior
Right renal vein is ____ than left.
shorter
Normal renal veins should demonstrate ____ flow and maybe pulsatile flow transmitted from ____.
phasic/ right atrium
______ renal arteries course through the hilum and are branches of the _____.
Segmental/ main renal artery
_____ arteries arise from segmentals and lie between the ______ and penetrate the _____.
Interlobar/ renal medullary pyraminds/ renal parenchyma
_____ arteries branch from the interlobars and turn at the cortico-medullary junction to course ____ to the cortex surface.
arcuate/ parallel
____ arteries extend into cortex.
Interlobar (cortical branches)
_____ renal arteries are common and may arise from ____, above or below the main renal arteries. On the right side, they may pass ____ to IVC. They may also arise from the ____ or _____.
Accessory (polar arteries)/ aorta/ anterior/ SMA, iliac arteries
Purpose of renal doppler exam
identify vascular etiology of hypertension/ prevent renal failure due to permanent parenchymal changes/ evaluate renal transplants for artery twists, kinks, stenosis, rejection, and renal vein thrombosis
Evaluates the entire renal artery, bilaterally, from the aorta to the renal parenchyma. Spectral waveforms obtained from the prox, mid, distal renal arteries and from segmental renal arteries. Limited by obesity, bowel gas, previous abdominal surgery, and prolonged exam time.
Direct color duplex renal artery exam
Examine only the segmental and/or interlobar arteries. Wavefrom characteristics within kidney are analyzed for abnormality associated with main renal artery stenosis or occlusion. Short exam time and high technical success rate, but limited to detecting severe stenoses.
Indirect color duplex renal artery exam
How to identify renal artery stenosis
Look for regions of velocity increase and post stenotic turbulence, carefully map these regions with spectral doppler/ record the peak systolic velocity from the waveform demonstrating max velocity/ calcualte the renal-aortic peak systolic velocity ration (RAR) from the highest velocity obtained in the renal artery with the velocity from the aorta
Normal Renal Artery characteristics
High flow velocity (PSV 100 +/- 20 cm/s)/ low resistance (RI < .75)/ high diastolic flow (EDV 30 +/- 5 cm/s)
Criteria for >60% Renal Artery Stenosis
Renal-aortic ratio > 3.5
Peak systolic velocity (PSV) > 180 cm/s
Post-stenotic turbulence
Low flow in distal renal artery
Renal Artery occlusion
No flow detectable in a well visualized renal artery/ low amplitude color and spectral doppler signal from the parenchyma/ small kidney size (<9 cm)
Renal parenchymal disease
blood flow in the renal artery of a normal kidney is of low resistance/ in the presence of renal parenchymal disease velocity is reduced and resistance is high in the main renal artery and in the segmental and interlobar arteries
Renal doppler physical limitations
excessive depth of renal arteries/ motion of respiration/ intra-abdominal gas/ obesity/ previous abdominal surgery
Technical limitations of Direct renal doppler
high technical failure rate/ accessory renal arteries occur in some pts and are difficult, if not impossible/ to find/ poor doppler angles/ long exam time/ requires high skill
Normal Indirect waveforms parameters
presence of an ESP (early systolic)/ Acceleration Time less than .07 sec/ presence of diastolic flow
Indirect criteria for > 60% stenosis
increased acceleration time/ tardus-parvus waveform/ loss of early systolic peak/ flattened systolic upslope/ acceleration <3 m/s/ abnormality lower ipsilateral RI/ reduced color flow in kidney unilaterally
Purpose of mesenteric doppler
diagnose mesenteric ischemia
very uncommon due to extensive collateral pathways/ usually caused by ather occlusive disease at vessel origins/ celiac, sma, and ima must all be involved for bowel ischemia to occur/ chronic or acute, usually caused by embolization
mesenteric ischemia
Mesenteric Ischemia symptoms
abdominal cramps and pain after eating/ diarrhea, change in bowel habits/ weight loss/ fear of food syndrome
Common hepatic artery branches
hepatic artery, gastroduodenal artery, pancreaticoduodenal artery
celiac axis branches
lt. gastric artery, splenic artery, common hepatic artery
supplies blood to the distal duodenum, small intestine, and colon
sma
arises from the aorta distal to renal arteries, supplies blood to the transverse descending, and sigmoid colong
ima
Celiac, hepatic, splenic arteries flow patterns
low resistance
sma & ima flow patterns
high resistance when fasting, low resistance post prandium
Criteria for >70% stenosis in mesenteric exam
Celiac artery: PSV > 200 cm/s with post stenotic turbulence
SMA: PSV > 275 cm/s with post stenotic turbulence
drains nutrient rich blood from the bowel and spleen into liver/ has capillary bed on each end of system, one it gut and other in parenchyma of liver
portal venous system
portal vein is formed by:
smv and splenic vein
portal vein course ____ in liver
intra-segmentally
Portal vein normal flow
low velocity (20-40 cm/s), continuous flow/ no filling defects with color doppler/ hepatopetal direction/ no varices/ normal flow direction in potential collaterals
elevated pressure in the portal venous system due to increased impedence of flow through the liver
portal hypertension
Causes of portal hypertension
prehepatic obstruction: thrombosis of portal or splenic vein
intrahepatic: cirrhosis (most common cause), hepatic fibrosis, lymphoma
post hepatic: ivc obstruction, hepatic vein obstruction
Most common cause of portal hypertension
cirrhosis
Portal hypertension can lead to:
ascites/ splenomegaly/ GI-esophageal bleeding/ jaundince/ signs of hepatic failure
Porto-systemic shunts
lt. gastric vein (coronary vein), gastric varices, recanalized paraumblical vein with hepatofugal flow, spleno-renal shunt
Gastic varices often occur:
near stomach (epigastrum), under lt lobe of liver, near the spleen
Spleno-renal vein
splenic vein to left renal vein
Transjugular Intrahepatic Portosystemic Shunt (TIPS)
stent placement in the liver parenchuma between the portal vein and hepatic vein/ purpose is decompression of the portal venous system/ does not address cause of portal hypertension
drains right hepatic lobe
right hepatic vein
lies between left and right hepatic lobes
middle hepatic vein
courses between medial and lateral segments of left lobe
left hepatic vein
hepatic veins enlarge as they approach....
the diaphragm
How are hepatic veins best imaged?
Trv, subcostally
stenosis or obstruction of hepatic veins, may be caused by hepatomegaly, splenomegaly, ascites
Budd-Chiari syndrome
Normal hepatic veins flow
respiratory variation, pulsatility