• Shuffle
    Toggle On
    Toggle Off
  • Alphabetize
    Toggle On
    Toggle Off
  • Front First
    Toggle On
    Toggle Off
  • Both Sides
    Toggle On
    Toggle Off
  • Read
    Toggle On
    Toggle Off
Reading...
Front

Card Range To Study

through

image

Play button

image

Play button

image

Progress

1/7

Click to flip

Use LEFT and RIGHT arrow keys to navigate between flashcards;

Use UP and DOWN arrow keys to flip the card;

H to show hint;

A reads text to speech;

7 Cards in this Set

  • Front
  • Back
A-131. What are the two types of muscle, their function, and fuel source?
Type 1: slow, sustained movement - many mitochondria and oxidatize enzymes

Type 2: fast, short duration movements - increased ATPase and glycogen
A-131. What is von Girke's Dz, what's missing, where does glycogen build up?
Type 1 metabolic myopathy

G6P deficient

Glycogen build up in the liver
A-131. What's lacking in Pompe's Dz and what are the S/S?
Lysosomal acid maltase deficiency (a-glucosidase)

Glycogen build up in liver, heart, muscle --> cardiomegaly, splenomegaly, muscle hypotonia
A-131. Cori's Dz, whats missing and the S/S?
Deficiency of Amyloglucosidase leading glycogen build up same as Pompes

Stunted growth, hepatomegaly, and hypoglycemia
A-131.Pts has glycogen building up in vesicles, in their muscles. What dz is it?
McArdle's
A-132. Path, presentation, dx, and tx of Myasthenia Gravis
Path: auto Abs block post-synaptic ACh receptors

Pres: muscle weakness worse with use

Dx: Tensilon screening test (edrophonium give and strenght increases)

Tx: Pyridostigmine (anticholinesterase)
A-132. How does Lambert-Eaton Syndrome differ from Myes. Gravis?
ABs towards presynaptic Ca Channels

Sx improve with muscle use and repeated muscle stimulation

Exacerbated by Aminoglycosides