Rickets is an extremely rare skeletal disease that softens and weakens the bones in children. When diagnosing rickets, the doctors usually see a delay in growth, bowing of the legs, weakness, and pain in the spine, pelvis and leg regions. Rickets develops when calcium cannot be absorbed so the body has to get the calcium from the bones which causes the bones to not mineralize. Rickets doesn’t discriminate; it affects children of all different races and genders. Rickets did not only affect the past, but still to this day it is affect children all around the world. During the 1940s, rickets was nearly eradicated because doctors found that the cause of rickets was vitamin D deficiency. Between 1896 and 2000 less than one third of children that
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Nutritional rickets is resulted from poor sunlight exposure. When a child doesn’t get the right amount of sunlight they don’t get enough vitamin D, and if they are replacing that with something that produces calcium it makes the bones brittle and weak. Usually Nutritional rickets appears in the first two years of a child’s life. Rickets is usually spotted in infants up to six months old upon seeing hypocalcemic tetany or seizures, while children that are older start to present with failure to thrive or skeletal deformities. The second type of rickets is Vitamin D Dependent. Vitamin D Dependent Type One is caused by a flaw in the genetic codes for the production of renal. Vitamin D Dependent Type Two is a really rare form of rickets. Type two is caused by an anomaly in the vitamin D receptors. Type two does not respond to any of the vitamin D treatments, but type one can be treated by giving a child a boost of vitamin D. The last rickets is different from the first two because it is passed down through generations. This form of rickets affects the X chromosome which causes hypophosphatemia sooner or later because of the renal wasting. There are many other causes of rickets like medical conditions or even the medication that the child is
Nutritional rickets is resulted from poor sunlight exposure. When a child doesn’t get the right amount of sunlight they don’t get enough vitamin D, and if they are replacing that with something that produces calcium it makes the bones brittle and weak. Usually Nutritional rickets appears in the first two years of a child’s life. Rickets is usually spotted in infants up to six months old upon seeing hypocalcemic tetany or seizures, while children that are older start to present with failure to thrive or skeletal deformities. The second type of rickets is Vitamin D Dependent. Vitamin D Dependent Type One is caused by a flaw in the genetic codes for the production of renal. Vitamin D Dependent Type Two is a really rare form of rickets. Type two is caused by an anomaly in the vitamin D receptors. Type two does not respond to any of the vitamin D treatments, but type one can be treated by giving a child a boost of vitamin D. The last rickets is different from the first two because it is passed down through generations. This form of rickets affects the X chromosome which causes hypophosphatemia sooner or later because of the renal wasting. There are many other causes of rickets like medical conditions or even the medication that the child is