Ms. Barth presents in a hyper-adrenergic state that causes acute pulmonary hypertension. Consequently, this causes pulmonary edema due to an increased hyperstatic pressure. Globally, she has systemic hypertension a result of vascular spasms and increased heart rate. Symptomatically she feels hot, sweaty, and anxious due to vasoconstriction (increasing heat retention) stimulation of sweat glands, and increased norepinephrine.
The cardiovascular system is dynamic and always changing to regulate blood flow to essential organs. Hypertension elevates the blood pressure within the ventricles which compresses the coronary vasculature, while on the other hand, downstream systemic vasculature and organs that receive the blood supply will be harmed as well. Consequently, ventricles will undergo hypertrophy due to a higher afterload, Vasculature elasticity will be compromised, and lastly highly perfused organs (heart, kidneys, and/or brain) will incur end-organ damage organs.
Hypertensions effects on the heart is very significant and progressive. For instance, to accommodate for a higher afterload the vascular wall must response to this pressure overload by increasing the wall thickness, inevitably causing concentric hypertrophy. Histologically cells haven’t grown in size but rather increased side to side …show more content…
However high systole pressure impedes this supply which can tear into the vascular layers causing vascular was to thicken and stiffen. Moreover, these arteries can narrow in effort to repair itself leading to coronary artery disease which compounds the effects of impeded blood flow. As this progresses myocytes may become abnormal leading to arrhythmias. Over time this strain can damage from heart attacks can cause your heart to weaken and your heart to become less efficient leading to heart
The cardiovascular system is dynamic and always changing to regulate blood flow to essential organs. Hypertension elevates the blood pressure within the ventricles which compresses the coronary vasculature, while on the other hand, downstream systemic vasculature and organs that receive the blood supply will be harmed as well. Consequently, ventricles will undergo hypertrophy due to a higher afterload, Vasculature elasticity will be compromised, and lastly highly perfused organs (heart, kidneys, and/or brain) will incur end-organ damage organs.
Hypertensions effects on the heart is very significant and progressive. For instance, to accommodate for a higher afterload the vascular wall must response to this pressure overload by increasing the wall thickness, inevitably causing concentric hypertrophy. Histologically cells haven’t grown in size but rather increased side to side …show more content…
However high systole pressure impedes this supply which can tear into the vascular layers causing vascular was to thicken and stiffen. Moreover, these arteries can narrow in effort to repair itself leading to coronary artery disease which compounds the effects of impeded blood flow. As this progresses myocytes may become abnormal leading to arrhythmias. Over time this strain can damage from heart attacks can cause your heart to weaken and your heart to become less efficient leading to heart