Implicit Memory

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"Implicit memory is shown when performing on a task is enabled in the absence of conscious remembrance; explicit memory is unmasked when the performance on a task requires conscious memory of previous occurrences (Graf and Schacter, 1985 in Anderson, 2015) and conveyed in typical tests of cued and free recall and recognition. Event memory loss after an injury is called anterograde amnesia which involves the extended hippocampal complex and the thalamus which helps form new memories (Salnaitis, 2015).
Comparing memory for an amnesic and normal individuals in the Graf and Schacter (1985) experiment, results show that the normal subject’s 68 % recall is significantly higher than the 5% in the amnesic subject, and the normal subject’s 34% is slightly higher than the amnesic subject’s 31% in fragment completion. This
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Amnesiac semantic memories are generally intact as their semantic comprehension is attained at an earlier time, and processes of encoding, consolidation for memory transfer, and reconsolidation occurs and data is stored into long-term memory, Additionally, both anterograde amnesia and retrograde may be present together and each can be different levels of amnesia and this cannot be explained (Salnaitis, 2015). However, Organic amnesia is memory loss caused from biological factors. “Patients with organic amnesia due to cerebral damage affecting medial temporal lobe structures, and related neural circuitry, show impaired performance on direct tests of memory for example, free recall, recognition, etc. Memory for recent data can also be studied by means of indirect measures of memory such as repetition priming. A large body of literature documents that patients with

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