cause by water intoxication due to renal water retention leading to hyponatremia, urine that is too concentrated when serum osmolarity is very high.
According to Huether & McCance (2012), Sign and symptom of SIADH depends on rate of development and severity of hyponetremia. In mild cases of hyponetremia, may or may not have any symptom. Norman sodium level in the blood is between 136 to 145 mEq/L. When this level drops from 140 to 130 mEq/L. some of the sign and symptoms may occur like increased thirst, impaired taste, poor appetite, short of breath on exertion, fatigue. When sodium level drops from 130 to 120 mEq/L, symptom like nausea vomiting or abdominal cramps might occur. Neurological symptom such as cause confusion, lethargy, muscle twitching, and convulsions, severe and sometimes irreversible neurologic damage may occur because of the cerebral edema when serum sodium level decrease below 110 to 115 mEq/L (p.449).
As Smeltzer and Bare (1996) explained, “Most of the …show more content…
According to Thomas (2015), “The following laboratory tests may be helpful in the diagnosis of SIADH: Serum sodium, potassium, chloride, and bicarbonate, plasma osmolality , serum creatinine, blood urea nitrogen, blood glucose, urine osmolality, serum uric acid, serum cortisol and thyroid-stimulating hormone. The patient’s volume should be assessed clinically to help rule out the presence of hypovolemia” (p.1). Further diagnostic imaging study to find the neurologic status like chest radiography to rule out pulmonary edema, computed tomography or magnetic resonance imaging of the head to for finding of cerebral edema is helpful. Medications that are known to cause hyponatremia by increasing secretion of ADH should be evaluated and discontinued. Evaluation of pain is necessary because pain or stress may stimulate limbic system to produce ADH (Thomas, C.,
According to Huether & McCance (2012), Sign and symptom of SIADH depends on rate of development and severity of hyponetremia. In mild cases of hyponetremia, may or may not have any symptom. Norman sodium level in the blood is between 136 to 145 mEq/L. When this level drops from 140 to 130 mEq/L. some of the sign and symptoms may occur like increased thirst, impaired taste, poor appetite, short of breath on exertion, fatigue. When sodium level drops from 130 to 120 mEq/L, symptom like nausea vomiting or abdominal cramps might occur. Neurological symptom such as cause confusion, lethargy, muscle twitching, and convulsions, severe and sometimes irreversible neurologic damage may occur because of the cerebral edema when serum sodium level decrease below 110 to 115 mEq/L (p.449).
As Smeltzer and Bare (1996) explained, “Most of the …show more content…
According to Thomas (2015), “The following laboratory tests may be helpful in the diagnosis of SIADH: Serum sodium, potassium, chloride, and bicarbonate, plasma osmolality , serum creatinine, blood urea nitrogen, blood glucose, urine osmolality, serum uric acid, serum cortisol and thyroid-stimulating hormone. The patient’s volume should be assessed clinically to help rule out the presence of hypovolemia” (p.1). Further diagnostic imaging study to find the neurologic status like chest radiography to rule out pulmonary edema, computed tomography or magnetic resonance imaging of the head to for finding of cerebral edema is helpful. Medications that are known to cause hyponatremia by increasing secretion of ADH should be evaluated and discontinued. Evaluation of pain is necessary because pain or stress may stimulate limbic system to produce ADH (Thomas, C.,