Hi Katrina, I enjoyed reading your poster presentation on gout and you have well described the symptoms and pathophysiology.
I did not know much about this topic and very interested to know that gout is a form of arthritis caused by monosodium urate crystal deposition in and around a joints(Vannuchi 2012). Clinically significant hyperuricemia is serum urate level > 6.8 mg/dL (404 mcmol/L) due to likelihood of urate crystal deposition when urate concentrations > 6.8 mg/dL (404 mcmol/L) (Burbage 2014).
Literature review shows that overall it is four times more common in men than women. Levels increase in women after menopause. Although the incidence of gout starts increasing from the age of 30 years, the peak prevalence and
I did not know much about this topic and very interested to know that gout is a form of arthritis caused by monosodium urate crystal deposition in and around a joints(Vannuchi 2012). Clinically significant hyperuricemia is serum urate level > 6.8 mg/dL (404 mcmol/L) due to likelihood of urate crystal deposition when urate concentrations > 6.8 mg/dL (404 mcmol/L) (Burbage 2014).
Literature review shows that overall it is four times more common in men than women. Levels increase in women after menopause. Although the incidence of gout starts increasing from the age of 30 years, the peak prevalence and