Day 1
Summary: John is a 62-year-old male smoker who has presented with concerning a BMI and blood test results.
Q1.
John’s low density glycoprotein levels are higher than the target level (actual level 5.6, target 1.0). Long term decreased high density glycoprotein level leads to a build-up of cholesterol in the body. Additionally, John’s triglyceride levels are above the recommended target level (actual level 5.7, target level <4.0). This supports the other data gathered, suggesting that John has a poor diet which is leading to a build-up of cholesterol in the body.
Q2.
There are both modifiable risks and non-modifiable risks which can lead to the development of atherosclerosis. Modifiable risks are those which can be altered such as lifestyle choices, whereas non-modifiable risks are those such …show more content…
Majority of John’s risk factors for developing atherosclerosis are modifiable risks. These include; obesity, sedentary lifestyle and being a smoker. There are also other factors which can be pieced together from the case or interpreted from results, these include; dyslipidaemia (from the blood test results), hypertension (from the elevated blood pressure), inflammation (considering other factors inflammation of the body is likely) and finally stress (mental (a friend’s death and physical (excessive weight i.e. stress on the bones and joints)). John unfortunately also two non-modifiable risks and these include his age (62 years old) and gender (male).
Q3.
There are four main stags of the progression of atherosclerosis, these are; endothelial cell injury and inflammation, fatty streak, hard/fibrous plaque and finally soft/complicated plaque. When an endothelial cell is injured this allows LDPs to enter the sub endothelium, this is likely to
Summary: John is a 62-year-old male smoker who has presented with concerning a BMI and blood test results.
Q1.
John’s low density glycoprotein levels are higher than the target level (actual level 5.6, target 1.0). Long term decreased high density glycoprotein level leads to a build-up of cholesterol in the body. Additionally, John’s triglyceride levels are above the recommended target level (actual level 5.7, target level <4.0). This supports the other data gathered, suggesting that John has a poor diet which is leading to a build-up of cholesterol in the body.
Q2.
There are both modifiable risks and non-modifiable risks which can lead to the development of atherosclerosis. Modifiable risks are those which can be altered such as lifestyle choices, whereas non-modifiable risks are those such …show more content…
Majority of John’s risk factors for developing atherosclerosis are modifiable risks. These include; obesity, sedentary lifestyle and being a smoker. There are also other factors which can be pieced together from the case or interpreted from results, these include; dyslipidaemia (from the blood test results), hypertension (from the elevated blood pressure), inflammation (considering other factors inflammation of the body is likely) and finally stress (mental (a friend’s death and physical (excessive weight i.e. stress on the bones and joints)). John unfortunately also two non-modifiable risks and these include his age (62 years old) and gender (male).
Q3.
There are four main stags of the progression of atherosclerosis, these are; endothelial cell injury and inflammation, fatty streak, hard/fibrous plaque and finally soft/complicated plaque. When an endothelial cell is injured this allows LDPs to enter the sub endothelium, this is likely to